Compounds > antimycin a
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antimycin a and epidermal growth factor

antimycin a has been researched along with epidermal growth factor in 5 studies

Research

Studies (5)

TimeframeStudies, this research(%)All Research%
pre-19902 (40.00)18.7374
1990's1 (20.00)18.2507
2000's2 (40.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Coulter, SJ; Hertel, C; Perkins, JP1
Hasegawa, T; Iwashima, A; Yamanishi, K1
Levine, JS; Lieberthal, W; Menza, SA1
Felts, N; Floyd, RA; Hensley, K; Pye, QN; Salsman, S1
Albano, A; Chen, K; Keaney, JF; Murphy, MP; Thomas, SR1

Other Studies

5 other study(ies) available for antimycin a and epidermal growth factor

ArticleYear
The involvement of cellular ATP in receptor-mediated internalization of epidermal growth factor and hormone-induced internalization of beta-adrenergic receptors.
    The Journal of biological chemistry, 1986, May-05, Volume: 261, Issue:13

    Topics: Adenosine Triphosphate; Antimycin A; Astrocytoma; Binding, Competitive; Cell Line; Deoxyglucose; Endocytosis; Epidermal Growth Factor; ErbB Receptors; Humans; Isoproterenol; Kinetics; Receptors, Adrenergic, beta; Receptors, Cell Surface

1986
Effects of vanadate on intracellular Ca2+ redistribution and hexose transport across plasma membrane in cultured mouse fibroblasts.
    FEBS letters, 1984, Aug-20, Volume: 174, Issue:1

    Topics: Animals; Antimycin A; Calcium; Calcium-Transporting ATPases; Carrier Proteins; Cell Membrane; Cells, Cultured; Deoxy Sugars; Deoxyglucose; Egtazic Acid; Epidermal Growth Factor; Fibroblasts; Kinetics; Mice; Monosaccharide Transport Proteins; Oligomycins; Vanadates; Vanadium

1984
Graded ATP depletion can cause necrosis or apoptosis of cultured mouse proximal tubular cells.
    The American journal of physiology, 1998, Volume: 274, Issue:2

    Topics: Adenosine Triphosphate; Animals; Antimycin A; Apoptosis; Cell Survival; Cells, Cultured; Deoxyglucose; DNA Fragmentation; Epidermal Growth Factor; Glucose; Insulin; Kidney Tubules, Proximal; Mice; Mice, Inbred C57BL; Microscopy, Electron; Microscopy, Fluorescence; Microscopy, Phase-Contrast; Necrosis

1998
Induction of Akt phosphorylation in rat primary astrocytes by H2O2 occurs upstream of phosphatidylinositol 3-kinase: no evidence for oxidative inhibition of PTEN.
    Archives of biochemistry and biophysics, 2001, Feb-15, Volume: 386, Issue:2

    Topics: Acetylcysteine; Androstadienes; Animals; Antimycin A; Astrocytes; Blotting, Western; Cells, Cultured; Epidermal Growth Factor; ErbB Receptors; Hydrogen Peroxide; Phosphatidylinositol 3-Kinases; Phosphatidylinositol 4,5-Diphosphate; Phosphoinositide-3 Kinase Inhibitors; Phosphoric Monoester Hydrolases; Phosphorylation; Phosphoserine; Protein Serine-Threonine Kinases; Proto-Oncogene Proteins; Proto-Oncogene Proteins c-akt; PTEN Phosphohydrolase; Rats; Rotenone; Signal Transduction; Tumor Suppressor Proteins; Uncoupling Agents; Wortmannin

2001
Mitochondrial function is required for hydrogen peroxide-induced growth factor receptor transactivation and downstream signaling.
    The Journal of biological chemistry, 2004, Aug-13, Volume: 279, Issue:33

    Topics: Animals; Antimycin A; Apoptosis; Blotting, Western; Carbonyl Cyanide m-Chlorophenyl Hydrazone; COS Cells; DNA, Mitochondrial; Enzyme Activation; Epidermal Growth Factor; Flow Cytometry; Hydrogen Peroxide; JNK Mitogen-Activated Protein Kinases; MAP Kinase Kinase 4; Mice; Mitochondria; Mitogen-Activated Protein Kinase Kinases; NIH 3T3 Cells; Oligomycins; Oxidative Stress; Potassium Cyanide; Precipitin Tests; Receptors, Growth Factor; Receptors, Platelet-Derived Growth Factor; Receptors, Vascular Endothelial Growth Factor; Rotenone; Signal Transduction; Transcriptional Activation; Tumor Necrosis Factor-alpha; Ultraviolet Rays

2004