Compounds > angiotensin ii
Page last updated: 2024-08-22

angiotensin ii and tubastatin a

angiotensin ii has been researched along with tubastatin a in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's3 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Cavasin, MA; Chen, B; Demos-Davies, KM; Ferguson, BS; Ferrara, C; Horn, TR; Jeong, MY; Mahaffey, JH; McKinsey, TA; Piroddi, N; Poggesi, C; Scellini, B; Schuetze, KB; Spiltoir, JI; Tesi, C; Williams, SM1
Cho, JY; Cho, SN; Choi, SY; Jeong, MH; Kee, HJ; Kim, GR; Kim, HS; Kim, IK; Ryu, Y1
Berkowitz, DE; Byeon, HE; Chi, Z; Choi, J; Jeong, Y; Kim, JH; Lee, SY; Lee, W; Nguyen, QT; Pandey, D; Seo, E1

Other Studies

3 other study(ies) available for angiotensin ii and tubastatin a

ArticleYear
HDAC6 contributes to pathological responses of heart and skeletal muscle to chronic angiotensin-II signaling.
    American journal of physiology. Heart and circulatory physiology, 2014, Jul-15, Volume: 307, Issue:2

    Topics: Angiotensin II; Animals; Cardiomegaly; Disease Models, Animal; Fibrosis; Heart Failure; Histone Deacetylase 6; Histone Deacetylase Inhibitors; Histone Deacetylases; Hydroxamic Acids; Indoles; Male; Mice; Mice, Knockout; Muscle, Skeletal; Muscular Atrophy; Myocardium; Signal Transduction; Stroke Volume; Systole; Time Factors; Ventricular Function, Left; Ventricular Remodeling

2014
Tubastatin A suppresses renal fibrosis via regulation of epigenetic histone modification and Smad3-dependent fibrotic genes.
    Vascular pharmacology, 2015, Volume: 72

    Topics: Angiotensin II; Animals; Cells, Cultured; Collagen Type I; Epigenesis, Genetic; Fibrosis; Histone Deacetylase 6; Histone Deacetylase Inhibitors; Histone Deacetylases; Histones; Hydroxamic Acids; Indoles; Inflammation; Kidney; Male; Mice; Mice, Inbred ICR; Signal Transduction; Smad2 Protein; Smad3 Protein; Transforming Growth Factor beta

2015
Histone deacetylase 6 inhibitor tubastatin A attenuates angiotensin II-induced hypertension by preventing cystathionine γ-lyase protein degradation.
    Pharmacological research, 2019, Volume: 146

    Topics: Angiotensin II; Animals; Aorta; Cystathionine gamma-Lyase; Endothelial Cells; HEK293 Cells; Histone Deacetylase 6; Histone Deacetylase Inhibitors; Humans; Hydrogen Sulfide; Hydroxamic Acids; Hypertension; Indoles; Male; Mice, Inbred C57BL; Proteolysis

2019