Page last updated: 2024-08-21

aminoacetonitrile and naloxone

aminoacetonitrile has been researched along with naloxone in 9 studies

Research

Studies (9)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's6 (66.67)29.6817
2010's3 (33.33)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Almela, P; Laorden, ML; Milanés, MV1
Díaz, MF; Gutstein, HB; Moulédous, L1
Almela, P; Laorden, M; Milanés, M1
Castells, MT; Laorden, ML; Milanés, MV; Núñez, C1
Almela, P; Atucha, NM; Laorden, ML; Milanés, MV1
Bryant, CD; Eitan, S; Evans, CJ; Hodgson, SR; Hofford, RS; Roberts, KW1
Almela, P; Atucha, NM; Laorden, ML; Martínez-Laorden, E; Milanés, MV1
Almela, P; Hurle, MA; Laorden, ML; Martínez-Laorden, E; Milanés, MV1
Calza, A; Ciccarelli, A; Concas, A; Giustetto, M; Grasso, F; Santoru, F; Sassoè-Pognetto, M1

Other Studies

9 other study(ies) available for aminoacetonitrile and naloxone

ArticleYear
Activation of the ERK signalling pathway contributes to the adaptive changes in rat hearts during naloxone-induced morphine withdrawal.
    British journal of pharmacology, 2007, Volume: 151, Issue:6

    Topics: Aminoacetonitrile; Animals; Blotting, Western; Extracellular Signal-Regulated MAP Kinases; Gene Expression; Genes, fos; Heart Ventricles; Immunohistochemistry; Male; MAP Kinase Signaling System; Morphine; Naloxone; Narcotic Antagonists; Narcotics; Phosphorylation; Rats; Rats, Sprague-Dawley; Signal Transduction; Substance Withdrawal Syndrome

2007
Extracellular signal-regulated kinase (ERK) inhibition does not prevent the development or expression of tolerance to and dependence on morphine in the mouse.
    Pharmacology, biochemistry, and behavior, 2007, Volume: 88, Issue:1

    Topics: Aminoacetonitrile; Animals; Blotting, Western; Dose-Response Relationship, Drug; Drug Tolerance; Enzyme Inhibitors; Extracellular Signal-Regulated MAP Kinases; Male; Mice; Mice, Inbred C57BL; Mitogen-Activated Protein Kinase 1; Mitogen-Activated Protein Kinase 3; Morphine Dependence; Naloxone; Narcotic Antagonists; Neuronal Plasticity; Pain Measurement; Reaction Time; Substance Withdrawal Syndrome

2007
The PKs PKA and ERK 1/2 are involved in phosphorylation of TH at Serine 40 and 31 during morphine withdrawal in rat hearts.
    British journal of pharmacology, 2008, Volume: 155, Issue:1

    Topics: Aminoacetonitrile; Animals; Cyclic AMP-Dependent Protein Kinases; Disease Models, Animal; Drug Implants; Isoquinolines; Male; Mitogen-Activated Protein Kinase 1; Mitogen-Activated Protein Kinase 3; Morphine; Morphine Dependence; Myocardium; Naloxone; Naphthalenes; Narcotic Antagonists; Norepinephrine; Phosphorylation; Protein Kinase C-delta; Protein Kinase Inhibitors; Rats; Rats, Sprague-Dawley; Serine; Substance Withdrawal Syndrome; Sulfonamides; Tyrosine 3-Monooxygenase

2008
Regulation of extracellular signal-regulated kinases (ERKs) by naloxone-induced morphine withdrawal in the brain stress system.
    Naunyn-Schmiedeberg's archives of pharmacology, 2008, Volume: 378, Issue:4

    Topics: Aminoacetonitrile; Animals; Blotting, Western; Brain; Corticotropin-Releasing Hormone; Extracellular Signal-Regulated MAP Kinases; Immunochemistry; Injections, Subcutaneous; Male; Morphine; Morphine Dependence; Naloxone; Narcotic Antagonists; Narcotics; Paraventricular Hypothalamic Nucleus; Protease Inhibitors; Proto-Oncogene Proteins c-fos; Rats; Rats, Sprague-Dawley; Signal Transduction; Solitary Nucleus; Substance Withdrawal Syndrome; Time Factors

2008
Cross-talk between protein kinase A and mitogen-activated protein kinases signalling in the adaptive changes observed during morphine withdrawal in the heart.
    The Journal of pharmacology and experimental therapeutics, 2009, Volume: 330, Issue:3

    Topics: Adaptation, Physiological; Aminoacetonitrile; Analgesics, Opioid; Animals; Blotting, Western; Cyclic AMP-Dependent Protein Kinases; Enzyme Inhibitors; Heart; Hemodynamics; Isoquinolines; Male; Mitogen-Activated Protein Kinases; Morphine; Naloxone; Naphthalenes; Narcotic Antagonists; Phosphorylation; Protein Kinase C; Rats; Rats, Sprague-Dawley; Signal Transduction; Substance Withdrawal Syndrome; Sulfonamides; Tyrosine 3-Monooxygenase; Weight Gain

2009
Extracellular signal-regulated kinase activation in the amygdala mediates elevated plus maze behavior during opioid withdrawal.
    Behavioural pharmacology, 2009, Volume: 20, Issue:7

    Topics: Aminoacetonitrile; Amygdala; Animals; Enzyme Activation; Extracellular Signal-Regulated MAP Kinases; Male; Maze Learning; Mice; Mice, Inbred C57BL; Morphine; Naloxone; Septum of Brain; Substance Withdrawal Syndrome

2009
Naloxone-precipitated morphine withdrawal evokes phosphorylation of heat shock protein 27 in rat heart through extracellular signal-regulated kinase.
    Journal of molecular and cellular cardiology, 2011, Volume: 51, Issue:1

    Topics: Aminoacetonitrile; Animals; Extracellular Signal-Regulated MAP Kinases; Heart; Heart Ventricles; HSP27 Heat-Shock Proteins; Immunoblotting; Morphine; Morphine Dependence; Myocardium; Naloxone; Phosphorylation; Protease Inhibitors; Rats; Rats, Sprague-Dawley; Substance Withdrawal Syndrome

2011
Morphine withdrawal activates hypothalamic-pituitary-adrenal axis and heat shock protein 27 in the left ventricle: the role of extracellular signal-regulated kinase.
    The Journal of pharmacology and experimental therapeutics, 2012, Volume: 342, Issue:3

    Topics: Adrenergic beta-Antagonists; Adrenocorticotropic Hormone; Aminoacetonitrile; Animals; Catechol O-Methyltransferase; Corticosterone; Extracellular Signal-Regulated MAP Kinases; Heart; Heart Ventricles; HSP27 Heat-Shock Proteins; Hypothalamo-Hypophyseal System; Male; Morphine; Morphine Dependence; Naloxone; Norepinephrine; Phosphorylation; Pituitary-Adrenal System; Propranolol; Rats; Rats, Sprague-Dawley; Receptors, Adrenergic, beta; Substance Withdrawal Syndrome

2012
Morphine withdrawal produces ERK-dependent and ERK-independent epigenetic marks in neurons of the nucleus accumbens and lateral septum.
    Neuropharmacology, 2013, Volume: 70

    Topics: Acetylation; Aminoacetonitrile; Animals; Cytoskeletal Proteins; Epigenesis, Genetic; Gene Expression; Histones; Male; MAP Kinase Signaling System; Methyl-CpG-Binding Protein 2; Morphine; Naloxone; Nerve Tissue Proteins; Neurons; Nucleus Accumbens; Phosphorylation; Proto-Oncogene Proteins c-fos; Rats; Septal Nuclei; Signal Transduction; Substance Withdrawal Syndrome

2013