aminoacetonitrile has been researched along with corticosterone in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 3 (100.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Almela, P; Hurle, MA; Laorden, ML; Martínez-Laorden, E; Milanés, MV | 1 |
| Chen, SM; Chen, XM; Geng, D; Li, CF; Liu, Q; Mu, RH; Wang, SS; Yi, LT | 1 |
| Eun, PH; Gonzales, EL; Kang, RJ; Kim, DG; Kim, JW; Kim, Y; Ko, MJ; Oh, HA; Seung, H; Shin, CY | 1 |
3 other study(ies) available for aminoacetonitrile and corticosterone
| Article | Year |
|---|---|
Morphine withdrawal activates hypothalamic-pituitary-adrenal axis and heat shock protein 27 in the left ventricle: the role of extracellular signal-regulated kinase.
Topics: Adrenergic beta-Antagonists; Adrenocorticotropic Hormone; Aminoacetonitrile; Animals; Catechol O-Methyltransferase; Corticosterone; Extracellular Signal-Regulated MAP Kinases; Heart; Heart Ventricles; HSP27 Heat-Shock Proteins; Hypothalamo-Hypophyseal System; Male; Morphine; Morphine Dependence; Naloxone; Norepinephrine; Phosphorylation; Pituitary-Adrenal System; Propranolol; Rats; Rats, Sprague-Dawley; Receptors, Adrenergic, beta; Substance Withdrawal Syndrome | 2012 |
ERK-dependent brain-derived neurotrophic factor regulation by hesperidin in mice exposed to chronic mild stress.
Topics: Aminoacetonitrile; Animals; Antidepressive Agents; Brain-Derived Neurotrophic Factor; Corticosterone; Disease Models, Animal; Extracellular Signal-Regulated MAP Kinases; Food Deprivation; Food Preferences; Gene Expression Regulation; Hesperidin; Hippocampus; Male; Mice; Mice, Inbred ICR; Phosphorylation; Protease Inhibitors; Stress, Psychological; Sucrose | 2016 |
Social support rescues acute stress-induced cognitive impairments by modulating ERK1/2 phosphorylation in adolescent mice.
Topics: Age Factors; Aminoacetonitrile; Animal Communication; Animals; Cognitive Dysfunction; Corticosterone; Early Growth Response Protein 1; Male; MAP Kinase Signaling System; Maze Learning; Memory, Short-Term; Mice; Mice, Inbred ICR; Mitogen-Activated Protein Kinase 1; Mitogen-Activated Protein Kinase 3; Phosphorylation; Prefrontal Cortex; Protease Inhibitors; Social Behavior; Stress, Psychological; Transcriptional Activation; Up-Regulation | 2018 |