Page last updated: 2024-08-16

ag 1879 and gw-5074

ag 1879 has been researched along with gw-5074 in 2 studies

Research

Studies (2)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	0 (0.00)	29.6817
2010's	2 (100.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Kabir, M; Kerns, E; Nguyen, K; Shah, P; Sun, H; Wang, Y; Xu, X; Yu, KR	1
Bunaciu, RP; Jensen, HA; Varner, JD; Yen, A	1

Other Studies

2 other study(ies) available for ag 1879 and gw-5074

Article	Year
Predictive models of aqueous solubility of organic compounds built on A large dataset of high integrity. Bioorganic & medicinal chemistry, 2019, 07-15, Volume: 27, Issue:14 Topics: Drug Discovery; Organic Chemicals; Pharmaceutical Preparations; Solubility	2019
GW5074 and PP2 kinase inhibitors implicate nontraditional c-Raf and Lyn function as drivers of retinoic acid-induced maturation. Cellular signalling, 2015, Volume: 27, Issue:8 Topics: Antineoplastic Agents; Antineoplastic Combined Chemotherapy Protocols; CD11b Antigen; Cell Differentiation; Drug Resistance, Neoplasm; G1 Phase Cell Cycle Checkpoints; HL-60 Cells; Humans; Indoles; Leukemia, Promyelocytic, Acute; Molecular Targeted Therapy; Phenols; Phosphatidylinositol 3-Kinase; Phosphoinositide-3 Kinase Inhibitors; Protein Kinase Inhibitors; Proto-Oncogene Proteins; Proto-Oncogene Proteins c-raf; Pyrimidines; Signal Transduction; src-Family Kinases; Time Factors; Tretinoin	2015

Article

Year

Predictive models of aqueous solubility of organic compounds built on A large dataset of high integrity.

Bioorganic & medicinal chemistry, 2019, 07-15, Volume: 27, Issue:14

Topics: Drug Discovery; Organic Chemicals; Pharmaceutical Preparations; Solubility

2019

GW5074 and PP2 kinase inhibitors implicate nontraditional c-Raf and Lyn function as drivers of retinoic acid-induced maturation.

Cellular signalling, 2015, Volume: 27, Issue:8

Topics: Antineoplastic Agents; Antineoplastic Combined Chemotherapy Protocols; CD11b Antigen; Cell Differentiation; Drug Resistance, Neoplasm; G1 Phase Cell Cycle Checkpoints; HL-60 Cells; Humans; Indoles; Leukemia, Promyelocytic, Acute; Molecular Targeted Therapy; Phenols; Phosphatidylinositol 3-Kinase; Phosphoinositide-3 Kinase Inhibitors; Protein Kinase Inhibitors; Proto-Oncogene Proteins; Proto-Oncogene Proteins c-raf; Pyrimidines; Signal Transduction; src-Family Kinases; Time Factors; Tretinoin

2015