activins has been researched along with metallothionein in 4 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 1 (25.00) | 18.2507 |
| 2000's | 1 (25.00) | 29.6817 |
| 2010's | 1 (25.00) | 24.3611 |
| 2020's | 1 (25.00) | 2.80 |
| Authors | Studies |
|---|---|
| Boime, I; Byrne, MC; Kumar, TR; Matzuk, MM; Palapattu, G; Wang, P; Woodruff, TK | 1 |
| Chen, L; Cipriano, SC; Kumar, TR; Matzuk, MM | 1 |
| Demczuk, M; Golebiowski, A; Kilen, SM; Kipp, JL; Mayo, KE; Rodriguez, G | 1 |
| Chen, CW; Chen, YC; Fan, CK; Huang, HM; Huang, NK; Lee, YL; Liu, CW | 1 |
4 other study(ies) available for activins and metallothionein
| Article | Year |
|---|---|
Transgenic models to study gonadotropin function: the role of follicle-stimulating hormone in gonadal growth and tumorigenesis.
Topics: Activins; Animals; Crosses, Genetic; Female; Follicle Stimulating Hormone; Gene Expression Regulation; Hemorrhage; Homozygote; Humans; Infertility, Female; Infertility, Male; Inhibins; Male; Metallothionein; Mice; Mice, Mutant Strains; Mice, Transgenic; Oligopeptides; Ovarian Neoplasms; Ovary; Peptides; Polycystic Ovary Syndrome; Seminal Vesicles; Steroids; Testicular Neoplasms; Urinary Tract; Wasting Syndrome | 1999 |
Follistatin is a modulator of gonadal tumor progression and the activin-induced wasting syndrome in inhibin-deficient mice.
Topics: Activins; Animals; Disease Progression; Female; Follistatin; Glycoproteins; Inhibins; Liver Diseases; Male; Metallothionein; Mice; Mice, Transgenic; Ovarian Neoplasms; Peptides; Stomach Diseases; Survival Analysis; Testicular Neoplasms; Wasting Syndrome | 2000 |
Gene expression profiling reveals Cyp26b1 to be an activin regulated gene involved in ovarian granulosa cell proliferation.
Topics: Activins; Animals; Cell Proliferation; Cytochrome P-450 Enzyme Inhibitors; Cytochrome P-450 Enzyme System; Female; Gene Expression Regulation; Granulosa Cells; Male; Metallothionein; Mice; Mice, Transgenic; Retinoic Acid 4-Hydroxylase; RNA, Messenger; Tretinoin | 2011 |
Activin A downregulates the CD69-MT2A axis via p38MAPK to induce erythroid differentiation that sensitizes BCR-ABL-positive cells to imatinib.
Topics: Activins; Antigens, CD; Antigens, Differentiation, T-Lymphocyte; Apoptosis; Cell Differentiation; Drug Resistance, Neoplasm; Fusion Proteins, bcr-abl; Humans; Imatinib Mesylate; K562 Cells; Lectins, C-Type; Leukemia, Myelogenous, Chronic, BCR-ABL Positive; Metallothionein; p38 Mitogen-Activated Protein Kinases | 2022 |