acetylglucosamine has been researched along with transforming growth factor beta in 6 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 2 (33.33) | 29.6817 |
| 2010's | 3 (50.00) | 24.3611 |
| 2020's | 1 (16.67) | 2.80 |
| Authors | Studies |
|---|---|
| Brownlee, M; Du, XL; Edelstein, D; Fantus, IG; Goldberg, H; Rossetti, L; Wu, J; Ziyadeh, F | 1 |
| Breborowicz, A; Ciszewicz, M; Polubinska, A; Tam, P; Wu, G | 1 |
| Azuma, K; Imagawa, T; Minami, S; Okamoto, Y; Osaki, T; Tsuka, T; Wakuda, T | 1 |
| Fan, K; Li, N; Li, Z; Liu, X; Qi, J; Wang, L; Xu, H; Yin, P; Zha, X; Zhao, C | 1 |
| Chang, HH; Chen, YJ; Huang, CH; Yao, CC; Young, TH | 1 |
| Cho, JW; Ji, S; Kang, MJ; Kim, E; Kim, YJ; Kweon, TH; Park, YS; Yang, WH; Yi, EC | 1 |
6 other study(ies) available for acetylglucosamine and transforming growth factor beta
| Article | Year |
|---|---|
Hyperglycemia-induced mitochondrial superoxide overproduction activates the hexosamine pathway and induces plasminogen activator inhibitor-1 expression by increasing Sp1 glycosylation.
Topics: Acetylglucosamine; Animals; Cattle; Cells, Cultured; Glycosylation; Hexosamines; Hyperglycemia; Mitochondria; Phosphoserine; Phosphothreonine; Plasminogen Activator Inhibitor 1; Promoter Regions, Genetic; Sp1 Transcription Factor; Superoxides; Transforming Growth Factor beta | 2000 |
Changes in peritoneal mesothelial cells phenotype after chronic exposure to glucose or N-acetylglucosamine.
Topics: Acetylglucosamine; Animals; Anti-Inflammatory Agents; Biocompatible Materials; Cell Line; Cell Proliferation; Cells, Cultured; Cytokines; Dialysis Solutions; Drug Administration Schedule; Epithelial Cells; Fibronectins; Glucose; Humans; Hyaluronic Acid; Osmolar Concentration; Oxidative Stress; Peritoneal Cavity; Peritoneal Dialysis; Phenotype; Reactive Oxygen Species; Tissue Plasminogen Activator; Transforming Growth Factor beta; Vascular Endothelial Growth Factor A | 2007 |
Suppressive effects of N-acetyl-D-glucosamine on rheumatoid arthritis mouse models.
Topics: Acetylglucosamine; Animals; Arthritis, Experimental; Arthritis, Rheumatoid; Disease Models, Animal; Female; Glucosamine; Interleukin-10; Interleukin-2; Interleukin-6; Joints; Mice; Transforming Growth Factor beta; Tumor Necrosis Factor-alpha | 2012 |
Altered β1,6-GlcNAc branched N-glycans impair TGF-β-mediated epithelial-to-mesenchymal transition through Smad signalling pathway in human lung cancer.
Topics: Acetylglucosamine; Apoptosis; Blotting, Western; Carcinoma, Non-Small-Cell Lung; Cell Movement; Cell Proliferation; Cells, Cultured; Epithelial-Mesenchymal Transition; Flow Cytometry; Fluorescent Antibody Technique; HEK293 Cells; Humans; Immunoenzyme Techniques; Luciferases; Lung Neoplasms; Mesoderm; N-Acetylglucosaminyltransferases; Polysaccharides; Real-Time Polymerase Chain Reaction; Reverse Transcriptase Polymerase Chain Reaction; RNA, Messenger; Signal Transduction; Smad Proteins; Transforming Growth Factor beta | 2014 |
Hexosamine-Induced TGF-β Signaling and Osteogenic Differentiation of Dental Pulp Stem Cells Are Dependent on N-Acetylglucosaminyltransferase V.
Topics: Acetylglucosamine; Cell Differentiation; Cell Proliferation; Dental Pulp; Gene Expression Regulation, Developmental; Glucosamine; Hexosamines; Humans; N-Acetylglucosaminyltransferases; Osteogenesis; Protein Serine-Threonine Kinases; Receptor, Transforming Growth Factor-beta Type I; Receptors, Transforming Growth Factor beta; RNA, Messenger; Signal Transduction; Stem Cells; Transforming Growth Factor beta; Young Adult | 2015 |
O-GlcNAc stabilizes SMAD4 by inhibiting GSK-3β-mediated proteasomal degradation.
Topics: Acetylglucosamine; Breast Neoplasms; Female; Glycogen Synthase Kinase 3 beta; Humans; Lung Neoplasms; Protein Processing, Post-Translational; Proteolysis; Serine; Signal Transduction; Smad4 Protein; Threonine; Transforming Growth Factor beta; Tumor Cells, Cultured; Ubiquitin | 2020 |