Page last updated: 2024-08-22

acetylglucosamine and diethylnitrosamine

acetylglucosamine has been researched along with diethylnitrosamine in 2 studies

Research

Studies (2)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	1 (50.00)	18.2507
2000's	1 (50.00)	29.6817
2010's	0 (0.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Bhaumik, M; Guttenplan, J; Harris, T; Johnson, L; Rogler, C; Stanley, P; Sundaram, S	1
Rogler, CE; Stanley, P; Tang, J; Yang, X	1

Other Studies

2 other study(ies) available for acetylglucosamine and diethylnitrosamine

Article	Year
Progression of hepatic neoplasms is severely retarded in mice lacking the bisecting N-acetylglucosamine on N-glycans: evidence for a glycoprotein factor that facilitates hepatic tumor progression. Cancer research, 1998, Jul-01, Volume: 58, Issue:13 Topics: Acetylglucosamine; Animals; Carcinogens; Diethylnitrosamine; Disease Progression; Female; Genetic Vectors; Liver Neoplasms, Experimental; Male; Mice; N-Acetylglucosaminyltransferases; Neoplasm Proteins; Phenobarbital; Polysaccharides	1998
Reduced hepatocyte proliferation is the basis of retarded liver tumor progression and liver regeneration in mice lacking N-acetylglucosaminyltransferase III. Cancer research, 2003, Nov-15, Volume: 63, Issue:22 Topics: Acetylglucosamine; Animals; Apoptosis; Carbohydrate Sequence; Carcinogens; Cell Division; Diethylnitrosamine; Disease Progression; Female; Gene Expression Regulation, Neoplastic; Glycoproteins; Hepatectomy; Hepatocytes; Liver Neoplasms, Experimental; Liver Regeneration; Male; Mice; Mice, Inbred C57BL; Mice, Inbred CBA; Molecular Sequence Data; N-Acetylglucosaminyltransferases; Phenobarbital; Vascular Endothelial Growth Factor A	2003

Article

Year

Progression of hepatic neoplasms is severely retarded in mice lacking the bisecting N-acetylglucosamine on N-glycans: evidence for a glycoprotein factor that facilitates hepatic tumor progression.

Cancer research, 1998, Jul-01, Volume: 58, Issue:13

Topics: Acetylglucosamine; Animals; Carcinogens; Diethylnitrosamine; Disease Progression; Female; Genetic Vectors; Liver Neoplasms, Experimental; Male; Mice; N-Acetylglucosaminyltransferases; Neoplasm Proteins; Phenobarbital; Polysaccharides

1998

Reduced hepatocyte proliferation is the basis of retarded liver tumor progression and liver regeneration in mice lacking N-acetylglucosaminyltransferase III.

Cancer research, 2003, Nov-15, Volume: 63, Issue:22

Topics: Acetylglucosamine; Animals; Apoptosis; Carbohydrate Sequence; Carcinogens; Cell Division; Diethylnitrosamine; Disease Progression; Female; Gene Expression Regulation, Neoplastic; Glycoproteins; Hepatectomy; Hepatocytes; Liver Neoplasms, Experimental; Liver Regeneration; Male; Mice; Mice, Inbred C57BL; Mice, Inbred CBA; Molecular Sequence Data; N-Acetylglucosaminyltransferases; Phenobarbital; Vascular Endothelial Growth Factor A

2003