acetylcysteine has been researched along with butein in 4 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 3 (75.00) | 24.3611 |
2020's | 1 (25.00) | 2.80 |
Authors | Studies |
---|---|
Huh, G; Kang, K; Kim, DW; Lee, H; Lee, JW; Selvaraj, B | 1 |
Chang, WY; Choi, YH; Hyun, JW; Kim, GY; Kim, MO; Moon, DO | 1 |
Ho, YJ; Hsu, LS; Kao, SH; Lin, JF; Yang, LH; Yeh, CW | 1 |
Jang, JH; Kwon, TK; Min, KJ; Yang, ES | 1 |
4 other study(ies) available for acetylcysteine and butein
Article | Year |
---|---|
Synthesis and biological evaluation of isoliquiritigenin derivatives as a neuroprotective agent against glutamate mediated neurotoxicity in HT22 cells.
Topics: Animals; Cell Death; Cell Line; Chalcones; Dose-Response Relationship, Drug; Glutamic Acid; Mice; Molecular Structure; Neuroprotective Agents; Reactive Oxygen Species; Structure-Activity Relationship | 2020 |
Butein induces G(2)/M phase arrest and apoptosis in human hepatoma cancer cells through ROS generation.
Topics: Acetylcysteine; Anthracenes; Antineoplastic Agents; Antioxidants; Apoptosis; Carcinoma, Hepatocellular; Cell Cycle Proteins; Cell Division; Cell Proliferation; Cell Survival; Chalcones; Dose-Response Relationship, Drug; Enzyme Activation; G2 Phase; Glutathione; Hep G2 Cells; Humans; JNK Mitogen-Activated Protein Kinases; Liver Neoplasms; Phosphorylation; Protein Kinase Inhibitors; Reactive Oxygen Species | 2010 |
Butein inhibits the proliferation of breast cancer cells through generation of reactive oxygen species and modulation of ERK and p38 activities.
Topics: Acetylcysteine; Antioxidants; Breast Neoplasms; Caspase 3; Cell Line, Tumor; Cell Proliferation; Chalcones; Extracellular Signal-Regulated MAP Kinases; Female; Humans; p38 Mitogen-Activated Protein Kinases; Phosphorylation; Poly(ADP-ribose) Polymerases; Proto-Oncogene Proteins c-bcl-2; Reactive Oxygen Species | 2012 |
Inhibitory effect of butein on tumor necrosis factor-α-induced expression of cell adhesion molecules in human lung epithelial cells via inhibition of reactive oxygen species generation, NF-κB activation and Akt phosphorylation.
Topics: Acetylcysteine; Alveolar Epithelial Cells; Anti-Inflammatory Agents; Cell Adhesion; Cell Line; Cell Nucleus; Chalcones; Chromans; Gene Expression; Green Fluorescent Proteins; Humans; Intercellular Adhesion Molecule-1; MAP Kinase Signaling System; Mitogen-Activated Protein Kinases; Monocytes; NF-kappa B; Phosphorylation; Protein Processing, Post-Translational; Protein Transport; Proto-Oncogene Proteins c-akt; Reactive Oxygen Species; Tumor Necrosis Factor-alpha; Vascular Cell Adhesion Molecule-1 | 2012 |