acetylcysteine has been researched along with 3,4-dihydroxybenzohydroxamic acid in 2 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 2 (100.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Caslin, HL; Elford, HL; Kolawole, EM; McLeod, JJA; Paranjape, A; Qayum, AA; Ryan, JJ; Spence, AJ; Taruselli, MT | 1 |
| Caslin, HL; Elford, HL; Haque, TT; Kiwanuka, KN; Kolawole, EM; McLeod, JJA; Paranjape, A; Qayum, AA; Ryan, JJ; Spence, AJ; Taruselli, M | 1 |
2 other study(ies) available for acetylcysteine and 3,4-dihydroxybenzohydroxamic acid
| Article | Year |
|---|---|
Didox (3,4-dihydroxybenzohydroxamic acid) suppresses IL-33-induced cytokine production in primary mouse mast cells.
Topics: Acetylcysteine; Animals; Bone Marrow Cells; Chemokine CCL3; Female; Gene Expression Regulation; Genes, Reporter; Hydroxamic Acids; Hydroxyurea; Immunosuppressive Agents; Interleukin-13; Interleukin-33; Lipopolysaccharides; Luciferases; Male; Mast Cells; Mice; Mice, Inbred C57BL; NF-kappa B; Primary Cell Culture; Signal Transduction; Transcription Factor AP-1; Tumor Necrosis Factor-alpha | 2017 |
Didox (3,4-dihydroxybenzohydroxamic acid) suppresses IgE-mediated mast cell activation through attenuation of NFκB and AP-1 transcription.
Topics: Acetylcysteine; Animals; Anti-Inflammatory Agents; Antioxidants; Bone Marrow Cells; Catalase; Cell Degranulation; Cells, Cultured; Chemokine CCL3; Hydroxamic Acids; Hypersensitivity; Immunoglobulin E; Interleukin-13; Interleukin-6; Mast Cells; Mice; Mice, Inbred C57BL; NF-kappa B; Oxidative Stress; Reactive Oxygen Species; Superoxide Dismutase; Transcription Factor AP-1; Transcription, Genetic; Tumor Necrosis Factor-alpha | 2017 |