acetaminophen has been researched along with cyclin d1 in 6 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 2 (33.33) | 29.6817 |
2010's | 4 (66.67) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Aakre, ME; Chytil, A; Entingh, AJ; Law, BK; Moses, HL; Nørgaard, P; Waltner-Law, ME | 1 |
Brittingham, JA; Chiu, H; Dambach, DM; Durham, SK; Gardner, CR; Laskin, DL; Laskin, JD | 1 |
Kajander, H; Koskinen, ML; Tenhunen, J; Yang, R; Zhang, S; Zhu, S | 1 |
Cotoia, A; Oksala, N; Tenhunen, J; Yang, R; Zhang, S; Zhu, S | 1 |
Bi, H; Chen, P; Fan, X; Hou, X; Huang, M; Jiang, Y; Tan, H; Wang, Y; Zeng, H | 1 |
Apte, U; Bhushan, B; Manley, MW; Poudel, S; Roy, N | 1 |
6 other study(ies) available for acetaminophen and cyclin d1
Article | Year |
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Salicylate-induced growth arrest is associated with inhibition of p70s6k and down-regulation of c-myc, cyclin D1, cyclin A, and proliferating cell nuclear antigen.
Topics: Acetaminophen; Analgesics; Animals; Anti-Inflammatory Agents, Non-Steroidal; Aspirin; Cell Division; Cell Line; Cyclin A; Cyclin D1; Epidermal Growth Factor; Gene Expression Regulation; Genes, myc; Humans; Indomethacin; Insulin-Like Growth Factor I; Mice; Mitogen-Activated Protein Kinases; p38 Mitogen-Activated Protein Kinases; Proliferating Cell Nuclear Antigen; Proto-Oncogene Proteins c-myc; Ribosomal Protein S6 Kinases; Salicylates; Tetradecanoylphorbol Acetate | 2000 |
Role of tumor necrosis factor receptor 1 (p55) in hepatocyte proliferation during acetaminophen-induced toxicity in mice.
Topics: Acetaminophen; Analgesics, Non-Narcotic; Animals; Antigens, CD; CDC2-CDC28 Kinases; Cell Division; Cyclin A; Cyclin D1; Cyclin-Dependent Kinase 2; Cyclin-Dependent Kinase 4; Cyclin-Dependent Kinase Inhibitor p21; Cyclin-Dependent Kinases; Cyclins; DNA-Binding Proteins; Hepatocytes; Immunohistochemistry; Liver; Male; Mice; Mice, Inbred C57BL; Mice, Transgenic; Phosphatidylinositol 3-Kinases; Proto-Oncogene Proteins; Receptors, Tumor Necrosis Factor; Receptors, Tumor Necrosis Factor, Type I; Signal Transduction; STAT3 Transcription Factor; Trans-Activators; Up-Regulation | 2003 |
Ringer's lactate improves liver recovery in a murine model of acetaminophen toxicity.
Topics: Acetaminophen; Alanine Transaminase; Analgesics, Non-Narcotic; Animals; Aspartate Aminotransferases; Chemical and Drug Induced Liver Injury; Cyclin D1; Drug Overdose; Isotonic Solutions; Liver; Male; Mice; Mice, Inbred C57BL; NF-kappa B; Protein Binding; Ringer's Lactate; Tumor Necrosis Factor-alpha | 2011 |
High mobility group B1 impairs hepatocyte regeneration in acetaminophen hepatotoxicity.
Topics: Acetaminophen; Alanine Transaminase; Animals; Antibodies, Anti-Idiotypic; Aspartate Aminotransferases; Cell Proliferation; Chemical and Drug Induced Liver Injury; Cyclin D1; Dose-Response Relationship, Drug; Hepatocytes; HMGB1 Protein; Immunoglobulin G; Liver Regeneration; Male; Mice; Mice, Inbred C57BL; Models, Animal; NF-kappa B | 2012 |
Dynamic and coordinated regulation of KEAP1-NRF2-ARE and p53/p21 signaling pathways is associated with acetaminophen injury responsive liver regeneration.
Topics: Acetaminophen; Adaptor Proteins, Signal Transducing; Animals; Carboxylic Ester Hydrolases; Cell Proliferation; Chemical and Drug Induced Liver Injury; Cyclin D1; Cyclin-Dependent Kinase 4; Cyclin-Dependent Kinase Inhibitor p21; Cytoskeletal Proteins; Hepatocytes; Kelch-Like ECH-Associated Protein 1; Liver; Liver Regeneration; Male; Mice; Mice, Inbred C57BL; NF-E2-Related Factor 2; Proliferating Cell Nuclear Antigen; Signal Transduction; Tumor Suppressor Protein p53 | 2014 |
Inhibition of Glycogen Synthase Kinase 3 Accelerated Liver Regeneration after Acetaminophen-Induced Hepatotoxicity in Mice.
Topics: Acetaminophen; Animals; beta Catenin; Cell Proliferation; Chemical and Drug Induced Liver Injury; Cyclin D1; Drug Overdose; Glycogen Synthase Kinase 3; Hepatocytes; Liver Regeneration; Mice; Mice, Inbred C57BL; Oligopeptides; Proliferating Cell Nuclear Antigen; Protein Kinase Inhibitors | 2017 |