acetaminophen and 2-aminoethoxydiphenyl borate

acetaminophen has been researched along with 2-aminoethoxydiphenyl borate in 2 studies

Research

Studies (2)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	0 (0.00)	29.6817
2010's	2 (100.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Bohr, S; Iracheta-Velle, A; Iracheta-Vellve, A; Jindal, R; King, KR; Li, M; Milwid, JM; Parekkadan, B; Patel, SJ; Vitalo, A; Yarmush, ML	1
Du, K; Farhood, A; Jaeschke, H; McGill, MR; Vinken, M; Williams, CD; Xie, Y	1

Other Studies

2 other study(ies) available for acetaminophen and 2-aminoethoxydiphenyl borate

Article	Year
Gap junction inhibition prevents drug-induced liver toxicity and fulminant hepatic failure. Nature biotechnology, 2012, Jan-15, Volume: 30, Issue:2 Topics: Acetaminophen; Animals; Boron Compounds; Chemical and Drug Induced Liver Injury; Connexins; Gap Junction beta-1 Protein; Gap Junctions; HeLa Cells; Humans; Liver Failure, Acute; Mice; Mice, Inbred C57BL; Protective Agents; Thioacetamide	2012
The gap junction inhibitor 2-aminoethoxy-diphenyl-borate protects against acetaminophen hepatotoxicity by inhibiting cytochrome P450 enzymes and c-jun N-terminal kinase activation. Toxicology and applied pharmacology, 2013, Dec-15, Volume: 273, Issue:3 Topics: Acetaminophen; Animals; Boron Compounds; Chemical and Drug Induced Liver Injury; Connexins; Cytochrome P-450 Enzyme Inhibitors; Cytochrome P-450 Enzyme System; Dimethyl Sulfoxide; Gap Junction beta-1 Protein; Gap Junctions; Hepatocytes; JNK Mitogen-Activated Protein Kinases; Liver; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; Signal Transduction; tert-Butylhydroperoxide	2013

Article

Year

Gap junction inhibition prevents drug-induced liver toxicity and fulminant hepatic failure.

Nature biotechnology, 2012, Jan-15, Volume: 30, Issue:2

Topics: Acetaminophen; Animals; Boron Compounds; Chemical and Drug Induced Liver Injury; Connexins; Gap Junction beta-1 Protein; Gap Junctions; HeLa Cells; Humans; Liver Failure, Acute; Mice; Mice, Inbred C57BL; Protective Agents; Thioacetamide

2012

The gap junction inhibitor 2-aminoethoxy-diphenyl-borate protects against acetaminophen hepatotoxicity by inhibiting cytochrome P450 enzymes and c-jun N-terminal kinase activation.

Toxicology and applied pharmacology, 2013, Dec-15, Volume: 273, Issue:3

Topics: Acetaminophen; Animals; Boron Compounds; Chemical and Drug Induced Liver Injury; Connexins; Cytochrome P-450 Enzyme Inhibitors; Cytochrome P-450 Enzyme System; Dimethyl Sulfoxide; Gap Junction beta-1 Protein; Gap Junctions; Hepatocytes; JNK Mitogen-Activated Protein Kinases; Liver; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; Signal Transduction; tert-Butylhydroperoxide

2013