7-ketocholesterol has been researched along with nifedipine in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 3 (100.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Athias, A; Berthier, A; Bessède, G; Gambert, P; Laubriet, A; Lemaire-Ewing, S; Lizard, G; Monier, S; Néel, D; Pais de Barros, JP; Prunet, C | 1 |
| Ichikawa, H; Katada, K; Kokura, S; Kon, T; Manabe, H; Naito, Y; Nakabe, N; Shimozawa, M; Yoshida, N; Yoshikawa, T | 1 |
| Miyashita, Y; Murano, T; Sasaki, H; Shirai, K; Watanabe, F | 1 |
3 other study(ies) available for 7-ketocholesterol and nifedipine
| Article | Year |
|---|---|
Involvement of a calcium-dependent dephosphorylation of BAD associated with the localization of Trpc-1 within lipid rafts in 7-ketocholesterol-induced THP-1 cell apoptosis.
Topics: Apoptosis; bcl-Associated Death Protein; Calcineurin; Calcium; Calcium Channel Blockers; Calcium Channels; Carrier Proteins; Cell Membrane; Cells, Cultured; Genes, bcl-2; Humans; Ketocholesterols; Membrane Microdomains; Monocytes; Nifedipine; Phosphorylation; Serine; TRPC Cation Channels; Verapamil | 2004 |
Azelnidipine, a new calcium channel blocker, inhibits endothelial inflammatory response by reducing intracellular levels of reactive oxygen species.
Topics: Aorta; Azetidinecarboxylic Acid; Biphenyl Compounds; Calcium Channel Blockers; Cell Adhesion; Cell Membrane; Cyclic N-Oxides; Dihydropyridines; Dose-Response Relationship, Drug; Endothelial Cells; Fluoresceins; Free Radical Scavengers; Gene Expression Regulation; Humans; Hydrazines; Indicators and Reagents; Inflammation; Ketocholesterols; Monocytes; NF-kappa B p50 Subunit; Nifedipine; Picrates; Protein Transport; Reactive Oxygen Species; RNA, Messenger; Time Factors; Tumor Necrosis Factor-alpha; U937 Cells; Vascular Cell Adhesion Molecule-1 | 2006 |
Vascular smooth muscle cell apoptosis induced by 7-ketocholesterol was mediated via Ca2+ and inhibited by the calcium channel blocker nifedipine.
Topics: Apoptosis; Calcium; Calcium Channel Blockers; Caspase 3; Humans; In Situ Nick-End Labeling; Ketocholesterols; Muscle, Smooth, Vascular; Nifedipine | 2007 |