4-butyrolactone has been researched along with orlistat in 3 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 3 (100.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Dünisch, P; Ewald, C; Freitag, D; Grube, S; Kalff, R; Klausnitzer, M; Sakr, Y; Walter, J | 1 |
Gunter, JH; Lubik, AA; Nelson, CC; Pouwer, RH; Quinn, RJ; Sadowski, MC | 1 |
Albanell, J; Arenas, EJ; Beltran, A; García-Rocha, M; Gomis, RR; Guinovart, JJ; Guiu, M; Lluch, A; Planet, E; Rojo, F; Rovira, A; Salvatella, X; Samino, S; Slebe, F; Testoni, G; Vinaixa, M; Yanes, O | 1 |
3 other study(ies) available for 4-butyrolactone and orlistat
Article | Year |
---|---|
Overexpression of fatty acid synthase in human gliomas correlates with the WHO tumor grade and inhibition with Orlistat reduces cell viability and triggers apoptosis.
Topics: 4-Butyrolactone; Apoptosis; Autophagy; Brain; Brain Neoplasms; Caspase 3; Cell Line, Tumor; Cell Proliferation; Cell Survival; Cells, Cultured; Cerulenin; Dose-Response Relationship, Drug; Fatty Acid Synthase, Type I; Fatty Acid Synthesis Inhibitors; Glioblastoma; Glioma; Humans; Lactones; Neoplasm Grading; Orlistat; Tissue Culture Techniques | 2014 |
The fatty acid synthase inhibitor triclosan: repurposing an anti-microbial agent for targeting prostate cancer.
Topics: 3T3 Cells; 4-Butyrolactone; Animals; Anti-Infective Agents, Local; Antineoplastic Agents; Apoptosis; Cell Line, Tumor; Drug Repositioning; Fatty Acid Synthase, Type I; Fatty Acid Synthesis Inhibitors; G1 Phase Cell Cycle Checkpoints; Humans; Lactones; Lipid Metabolism; Male; Mice; Orlistat; Prostatic Neoplasms; Structure-Activity Relationship; Triclosan | 2014 |
FoxA and LIPG endothelial lipase control the uptake of extracellular lipids for breast cancer growth.
Topics: 4-Butyrolactone; Animals; Biological Transport; Breast Neoplasms; Cell Line, Tumor; Cell Movement; Cell Proliferation; Doxycycline; Enzyme Inhibitors; Female; Gene Expression Regulation, Neoplastic; Hepatocyte Nuclear Factor 3-alpha; Hepatocyte Nuclear Factor 3-beta; Humans; Lactones; Lipase; Lipid Metabolism; MCF-7 Cells; Mice; Mice, Nude; Neoplasm Invasiveness; Orlistat; RNA, Small Interfering; Signal Transduction; Xenograft Model Antitumor Assays | 2016 |