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4-(n-methyl-n-nitrosamino)-1-(3-pyridyl)-1-butanone and cucurbitacin b

4-(n-methyl-n-nitrosamino)-1-(3-pyridyl)-1-butanone has been researched along with cucurbitacin b in 2 studies

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's2 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Bora, HK; Farhan, M; Khan, S; Kumar, S; Maurya, R; Meeran, SM; Shukla, S; Sinha, S1
Khan, S; Kumar, S; Maurya, R; Meeran, SM; Mitra, K; Shukla, S; Singh, K; Sinha, S1

Other Studies

2 other study(ies) available for 4-(n-methyl-n-nitrosamino)-1-(3-pyridyl)-1-butanone and cucurbitacin b

ArticleYear
Cucurbitacin B Alters the Expression of Tumor-Related Genes by Epigenetic Modifications in NSCLC and Inhibits NNK-Induced Lung Tumorigenesis.
    Cancer prevention research (Philadelphia, Pa.), 2015, Volume: 8, Issue:6

    Topics: Animals; Apoptosis; Blotting, Southwestern; Blotting, Western; Carcinogens; Carcinoma, Non-Small-Cell Lung; Cell Cycle; Cell Proliferation; Cell Transformation, Neoplastic; Chromatin Immunoprecipitation; DNA Methylation; Epigenesis, Genetic; Female; Gene Expression Regulation, Neoplastic; Genes, Neoplasm; Histone Deacetylases; Humans; Immunoenzyme Techniques; Lung Neoplasms; Mice; Mice, Inbred A; Nitrosamines; Promoter Regions, Genetic; Real-Time Polymerase Chain Reaction; Reverse Transcriptase Polymerase Chain Reaction; RNA, Messenger; Triterpenes; Tumor Cells, Cultured

2015
Cucurbitacin B inhibits the stemness and metastatic abilities of NSCLC via downregulation of canonical Wnt/β-catenin signaling axis.
    Scientific reports, 2016, Feb-24, Volume: 6

    Topics: A549 Cells; Animals; beta Catenin; Blotting, Western; Carcinoma, Non-Small-Cell Lung; Cell Line, Tumor; Cell Movement; Cell Proliferation; Cell Transformation, Neoplastic; Down-Regulation; Epithelial-Mesenchymal Transition; Female; Human Umbilical Vein Endothelial Cells; Humans; Immunoprecipitation; Lung Neoplasms; Mice; Microscopy, Fluorescence; Neovascularization, Physiologic; Nitrosamines; RNA Interference; RNA, Small Interfering; Transforming Growth Factor beta1; Triterpenes; Wnt Signaling Pathway; Wnt3 Protein; Wnt3A Protein

2016