Page last updated: 2024-09-03

20-hydroxy-5,8,11,14-eicosatetraenoic acid and 6-ketoprostaglandin f1 alpha

20-hydroxy-5,8,11,14-eicosatetraenoic acid has been researched along with 6-ketoprostaglandin f1 alpha in 2 studies

Compound Research Comparison

Studies
(20-hydroxy-5,8,11,14-eicosatetraenoic acid)
Trials
(20-hydroxy-5,8,11,14-eicosatetraenoic acid)
Recent Studies (post-2010)
(20-hydroxy-5,8,11,14-eicosatetraenoic acid)
Studies
(6-ketoprostaglandin f1 alpha)
Trials
(6-ketoprostaglandin f1 alpha)
Recent Studies (post-2010) (6-ketoprostaglandin f1 alpha)
640132334,832294150

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's1 (50.00)18.2507
2000's1 (50.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Campbell, WB; Falck, JR; Kurian, JB; Pratt, PF; Reddy, KM1
Falck, JR; Frisbee, JC; Lombard, JH; Maier, KG; Roman, RJ1

Other Studies

2 other study(ies) available for 20-hydroxy-5,8,11,14-eicosatetraenoic acid and 6-ketoprostaglandin f1 alpha

ArticleYear
20-HETE relaxes bovine coronary arteries through the release of prostacyclin.
    Hypertension (Dallas, Tex. : 1979), 1998, Volume: 31, Issue:1 Pt 2

    Topics: 15-Hydroxy-11 alpha,9 alpha-(epoxymethano)prosta-5,13-dienoic Acid; 6-Ketoprostaglandin F1 alpha; Animals; Bradykinin; Cattle; Coronary Vessels; Epoprostenol; Hydroxyeicosatetraenoic Acids; In Vitro Techniques; Isometric Contraction; Muscle Tonus; Muscle, Smooth, Vascular; Vasodilation

1998
Integration of hypoxic dilation signaling pathways for skeletal muscle resistance arteries.
    American journal of physiology. Regulatory, integrative and comparative physiology, 2002, Volume: 283, Issue:2

    Topics: 6-Ketoprostaglandin F1 alpha; Animals; Arteries; Cyclooxygenase Inhibitors; Enzyme Inhibitors; Epoprostenol; Hydroxyeicosatetraenoic Acids; Hypoxia; In Vitro Techniques; Male; Muscle, Skeletal; Nitric Oxide; Nitric Oxide Synthase; Potassium Channel Blockers; Potassium Channels; Rats; Rats, Sprague-Dawley; Signal Transduction; Vascular Resistance; Vasodilation

2002