2-amino-5-phosphonovalerate has been researched along with charybdotoxin in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 1 (33.33) | 18.2507 |
| 2000's | 1 (33.33) | 29.6817 |
| 2010's | 1 (33.33) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| D'Angelo, E; De Filippi, G; Rossi, P; Taglietti, V | 1 |
| Gerich, FJ; Hepp, S; Müller, M | 1 |
| Bai, Y; Chen, L; Li, L; Qi, J; Qi, Z; Sun, K; Yang, R; Zhang, Z; Zhou, L | 1 |
3 other study(ies) available for 2-amino-5-phosphonovalerate and charybdotoxin
| Article | Year |
|---|---|
Ionic mechanism of electroresponsiveness in cerebellar granule cells implicates the action of a persistent sodium current.
Topics: 2-Amino-5-phosphonovalerate; 6-Cyano-7-nitroquinoxaline-2,3-dione; Action Potentials; Animals; Calcium; Calcium Channel Blockers; Calcium Channels; Cerebellum; Charybdotoxin; Electric Stimulation; Excitatory Amino Acid Antagonists; Nerve Fibers; omega-Conotoxin GVIA; Patch-Clamp Techniques; Peptides; Periodicity; Potassium; Potassium Channels; Rats; Rats, Wistar; Signal Transduction; Sodium; Sodium Channels; Synaptic Transmission; Tetraethylammonium; Tetrodotoxin | 1998 |
Sulfhydryl oxidation reduces hippocampal susceptibility to hypoxia-induced spreading depression by activating BK channels.
Topics: 6-Cyano-7-nitroquinoxaline-2,3-dione; Aniline Compounds; Animals; Calcium; Charybdotoxin; Cortical Spreading Depression; Dose-Response Relationship, Radiation; Drug Interactions; Electric Stimulation; Excitatory Amino Acid Antagonists; Excitatory Postsynaptic Potentials; Glutamine; Hippocampus; Hypoxia; In Vitro Techniques; Large-Conductance Calcium-Activated Potassium Channels; Membrane Potentials; Neurons; Nickel; Oxidation-Reduction; Patch-Clamp Techniques; Peptides; Potassium Channel Blockers; Potassium Channels, Calcium-Activated; Rats; Rats, Sprague-Dawley; Sulfhydryl Reagents; Tolbutamide; Valine; Xanthenes | 2005 |
Cerebroside-A provides potent neuroprotection after cerebral ischaemia through reducing glutamate release and Ca²⁺ influx of NMDA receptors.
Topics: 6-Cyano-7-nitroquinoxaline-2,3-dione; Analysis of Variance; Animals; Brain Ischemia; Calcium; Cerebral Infarction; Cerebrosides; Charybdotoxin; Disease Models, Animal; Dose-Response Relationship, Drug; Drug Interactions; Electric Stimulation; Excitatory Amino Acid Antagonists; Excitatory Postsynaptic Potentials; Glucose; Glutamic Acid; Hippocampus; Hypoxia; In Vitro Techniques; Infarction, Middle Cerebral Artery; Long-Term Potentiation; Male; Mice; Mice, Inbred C57BL; N-Methylaspartate; Neuroprotective Agents; Neurotoxins; Patch-Clamp Techniques; Rats; Rats, Sprague-Dawley; Receptors, N-Methyl-D-Aspartate; Tetrazolium Salts; Valine | 2012 |