Compounds > 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine

1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine and lead

1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine has been researched along with lead in 2 studies

Research

Studies (2)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	0 (0.00)	29.6817
2010's	1 (50.00)	24.3611
2020's	1 (50.00)	2.80

Authors

Authors	Studies
Chang, W; Chen, J; Li, F; Li, J; Li, X; Ye, F; Yuan, J	1
Ayyalasomayajula, N; Bandaru, LJM; Challa, S; Dixit, PK; Murumulla, L	1

Other Studies

2 other study(ies) available for 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine and lead

Article	Year
Cyclosporin A protects against Lead neurotoxicity through inhibiting mitochondrial permeability transition pore opening in nerve cells. Neurotoxicology, 2016, Volume: 57 Topics: 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine; Adenosine Triphosphate; Animals; Apoptosis Inducing Factor; Cell Line, Transformed; Cyclosporins; Cytochromes c; Humans; L-Lactate Dehydrogenase; Lead; Membrane Potential, Mitochondrial; Mitochondrial Membrane Transport Proteins; Mitochondrial Permeability Transition Pore; Neuroblastoma; Neurons; Neuroprotective Agents; Neurotoxins; PC12 Cells; Rats; Reactive Oxygen Species	2016
Defective mitophagy and induction of apoptosis by the depleted levels of PINK1 and Parkin in Pb and β-amyloid peptide induced toxicity. Toxicology mechanisms and methods, 2022, Volume: 32, Issue:8 Topics: 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine; Alzheimer Disease; Amyloid beta-Peptides; Apoptosis; Humans; Lead; Mitophagy; Neuroblastoma; Protein Kinases; Reactive Oxygen Species; Ubiquitin-Protein Ligases	2022

Article

Year

Cyclosporin A protects against Lead neurotoxicity through inhibiting mitochondrial permeability transition pore opening in nerve cells.

Neurotoxicology, 2016, Volume: 57

Topics: 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine; Adenosine Triphosphate; Animals; Apoptosis Inducing Factor; Cell Line, Transformed; Cyclosporins; Cytochromes c; Humans; L-Lactate Dehydrogenase; Lead; Membrane Potential, Mitochondrial; Mitochondrial Membrane Transport Proteins; Mitochondrial Permeability Transition Pore; Neuroblastoma; Neurons; Neuroprotective Agents; Neurotoxins; PC12 Cells; Rats; Reactive Oxygen Species

2016

Defective mitophagy and induction of apoptosis by the depleted levels of PINK1 and Parkin in Pb and β-amyloid peptide induced toxicity.

Toxicology mechanisms and methods, 2022, Volume: 32, Issue:8

Topics: 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine; Alzheimer Disease; Amyloid beta-Peptides; Apoptosis; Humans; Lead; Mitophagy; Neuroblastoma; Protein Kinases; Reactive Oxygen Species; Ubiquitin-Protein Ligases

2022