Page last updated: 2024-08-23

1-deoxynojirimycin and exendin (9-39)

1-deoxynojirimycin has been researched along with exendin (9-39) in 2 studies

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's2 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Aoyama, T; Fujiwara, H; Hattori, A; Iwasa, M; Kawamura, I; Kobayashi, H; Minatoguchi, S; Nishigaki, K; Shiraki, T; Sumi, S; Takemura, G; Ushikoshi, H; Yamada, Y; Yamaki, T; Yasuda, S1
Iwasa, M; Kanamori, H; Kawaguchi, T; Kawasaki, M; Mikami, A; Minatoguchi, S; Naruse, G; Nishigaki, K; Yamada, Y; Yoshida, A1

Other Studies

2 other study(ies) available for 1-deoxynojirimycin and exendin (9-39)

ArticleYear
Both stimulation of GLP-1 receptors and inhibition of glycogenolysis additively contribute to a protective effect of oral miglitol against ischaemia-reperfusion injury in rabbits.
    British journal of pharmacology, 2011, Volume: 164, Issue:1

    Topics: 1-Deoxynojirimycin; Administration, Oral; Animals; Blood Glucose; Blood Pressure; Drug Synergism; Glucagon-Like Peptide 1; Glucagon-Like Peptide-1 Receptor; Glycogenolysis; Heart; Heart Rate; Hypoglycemic Agents; Insulin; Male; Myocardial Infarction; Myocardial Reperfusion Injury; Myocardium; Peptide Fragments; Phosphatidylinositol 3-Kinases; Proto-Oncogene Proteins c-akt; Rabbits; Receptors, Glucagon

2011
The intestine responds to heart failure by enhanced mitochondrial fusion through glucagon-like peptide-1 signalling.
    Cardiovascular research, 2019, Nov-01, Volume: 115, Issue:13

    Topics: 1-Deoxynojirimycin; Animals; Cells, Cultured; Cyclic AMP-Dependent Protein Kinases; Disease Models, Animal; Dynamins; Enteroendocrine Cells; Glucagon-Like Peptide 1; Glucagon-Like Peptide-1 Receptor; Glycoside Hydrolase Inhibitors; GTP Phosphohydrolases; Heart Failure; Ileum; Incretins; Male; Membrane Proteins; Mitochondria, Heart; Mitochondrial Dynamics; Mitochondrial Proteins; Myocytes, Cardiac; Paracrine Communication; Peptide Fragments; Rats, Inbred Dahl; Rats, Sprague-Dawley; Signal Transduction; Sodium Chloride, Dietary; Ventricular Function, Left

2019