Compounds > 1,2-bis(2-aminophenoxy)ethane n,n,n',n'-tetraacetic acid acetoxymethyl ester

1,2-bis(2-aminophenoxy)ethane n,n,n',n'-tetraacetic acid acetoxymethyl ester and nad

1,2-bis(2-aminophenoxy)ethane n,n,n',n'-tetraacetic acid acetoxymethyl ester has been researched along with nad in 5 studies

Research

Studies (5)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's1 (20.00)18.2507
2000's2 (40.00)29.6817
2010's1 (20.00)24.3611
2020's1 (20.00)2.80

Authors

AuthorsStudies
Karczewski, JM; Noordhoek, J; Peters, JG1
Chan, SL; Culmsee, C; Guo, Z; Kruman, II; Kruman, Y; Mattson, MP; Penix, L1
Bentle, MS; Bey, EA; Boothman, DA; Reinicke, KE; Spitz, DR1
Bair, JS; Bey, EA; Boothman, DA; Dong, Y; Gao, J; Hergenrother, PJ; Huang, X; Kilgore, JA; Li, LS; Parkinson, EI; Patel, M; Wang, Y; Williams, NS1
Cui, P; He, Z; Li, A; Liu, H; Sun, X; Wang, Y; Yan, J; Yan, X; Yu, L; Zhang, J1

Other Studies

5 other study(ies) available for 1,2-bis(2-aminophenoxy)ethane n,n,n',n'-tetraacetic acid acetoxymethyl ester and nad

ArticleYear
Prevention of oxidant-induced cell death in Caco-2 colon carcinoma cells after inhibition of poly(ADP-ribose) polymerase and Ca2+ chelation: involvement of a common mechanism.
    Biochemical pharmacology, 1999, Jan-01, Volume: 57, Issue:1

    Topics: Adenosine Triphosphate; Benzamides; Caco-2 Cells; Calcium; Cell Death; Chelating Agents; DNA Damage; DNA, Neoplasm; Egtazic Acid; Glutathione; Humans; Hydrogen Peroxide; Kinetics; NAD; Naphthoquinones; Oxidants; Oxidative Stress; Poly(ADP-ribose) Polymerase Inhibitors; Vitamin K

1999
Homocysteine elicits a DNA damage response in neurons that promotes apoptosis and hypersensitivity to excitotoxicity.
    The Journal of neuroscience : the official journal of the Society for Neuroscience, 2000, Sep-15, Volume: 20, Issue:18

    Topics: Animals; Apoptosis; Benzamides; Calcium; Cells, Cultured; DNA; DNA Damage; Egtazic Acid; Enzyme Inhibitors; Excitatory Amino Acid Agonists; Hippocampus; Homocysteine; Intracellular Fluid; Kainic Acid; Membrane Potentials; Mitochondria; NAD; Neurons; Poly(ADP-ribose) Polymerase Inhibitors; Poly(ADP-ribose) Polymerases; Rats; Tumor Suppressor Protein p53

2000
Calcium-dependent modulation of poly(ADP-ribose) polymerase-1 alters cellular metabolism and DNA repair.
    The Journal of biological chemistry, 2006, Nov-03, Volume: 281, Issue:44

    Topics: Adenosine Triphosphate; Calcium; Cell Death; Chelating Agents; DNA; DNA Damage; DNA Repair; Egtazic Acid; Enzyme Activation; Humans; Hydrogen Peroxide; NAD; Naphthoquinones; Poly (ADP-Ribose) Polymerase-1; Poly(ADP-ribose) Polymerases; Time Factors

2006
An NQO1 substrate with potent antitumor activity that selectively kills by PARP1-induced programmed necrosis.
    Cancer research, 2012, Jun-15, Volume: 72, Issue:12

    Topics: Adenosine Triphosphate; Antineoplastic Agents; Calcium; Cell Line, Tumor; DNA Damage; Egtazic Acid; Humans; NAD; NAD(P)H Dehydrogenase (Quinone); Naphthoquinones; Necrosis; Neoplasms; Oxidation-Reduction; Poly (ADP-Ribose) Polymerase-1; Poly(ADP-ribose) Polymerases; Quinones; Reactive Oxygen Species; RNA Interference; RNA, Small Interfering

2012
Rapidly Inhibiting the Inflammatory Cytokine Storms and Restoring Cellular Homeostasis to Alleviate Sepsis by Blocking Pyroptosis and Mitochondrial Apoptosis Pathways.
    Advanced science (Weinheim, Baden-Wurttemberg, Germany), 2023, Volume: 10, Issue:14

    Topics: Apoptosis; Cytokine Release Syndrome; Homeostasis; Humans; Inflammasomes; Inflammation; Lipopolysaccharides; NAD; NLR Family, Pyrin Domain-Containing 3 Protein; Pyroptosis; Sepsis

2023