A malabsorption syndrome that is precipitated by the ingestion of foods containing GLUTEN, such as wheat, rye, and barley. It is characterized by INFLAMMATION of the SMALL INTESTINE, loss of MICROVILLI structure, failed INTESTINAL ABSORPTION, and MALNUTRITION.
| Excerpt | Reference |
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| "Untreated celiac disease is characterized by gastrointestinal endocrine cell hyperplasia (ECH)." | ( Moyana, TN; Shukoor, S, 1991) |
| "Celiac disease is a multisystem disorder in which extraintestinal involvement is common." | ( Costello, FC; Faizallah, R; Lee, FI; Walker, R, 1982) |
| "Untreated celiac disease is characterized by high levels of 1,25-vitamin D and by increased bone turnover, caused by the increase in intact parathyroid hormone level." | ( Bernardi, M; Cecchetti, L; Corazza, GR; Corrao, G; Di Sario, A; Gasbarrini, G; Tarozzi, C, 1995) |
| "Celiac disease is frequently diagnosed in patients with nonspecific abdominal symptoms." | ( Ferenci, P; Genser, D; Granditsch, G; Lochs, H; Penner, E; Vogelsang, H; Wyatt, J, 1995) |
| "Adult celiac disease is associated with osteopenia, which is not always reversible after gluten-free diet (GFD)." | ( Cecchetti, L; Corazza, GR; Di Stefano, M; Gasbarrini, G; Jorizzo, RA; Minguzzi, L, 1997) |
| "Celiac disease is characterized by severe inflammation of the small intestine, and inflammation is known to often be associated with enhanced nitric oxide (NO) production." | ( Arends, JW; Buurman, W; Forget, P; ter Steege, J, 1997) |
| "Celiac disease is characterized by morphologic and functional aberrations of the small intestinal mucosa, i." | ( Fälth-Magnusson, K; Laurin, P; Magnusson, KE; Stenhammar, L; Sundqvist, T, 1998) |
| "Celiac disease is characterized by disturbed jejunal crypt-villus axis biology with immunoglobulin (Ig) A deposits underlining the epithelium." | ( Halttunen, T; Mäki, M, 1999) |
| "Celiac disease is a major cause of intestinal malabsorption." | ( Adams, JE; Davies, M; Mawer, EB; Selby, PL, 1999) |
| "Celiac disease is well documented especially from northern parts of India." | ( Thapa, BR, 1999) |
| "Celiac disease is a genetically-based intolerance to gluten." | ( Mantovani, A; Stazi, AV, 2000) |
| "Celiac disease is an HLA-DQ2-associated disorder characterized by intestinal T cell responses to ingested wheat gliadins." | ( Arentz-Hansen, H; Kett, K; Kristiansen, C; Lundin, KE; McAdam, S; Molberg, O; Sollid, LM, 2001) |
| "Celiac disease is an autoimmune disease with the damage of the intestinal barrier." | ( Kohout, P, 2001) |
| "The pathogenesis of celiac disease is not completely understood but, although the initial step of the process is still unclear, an altered immune response seems to play a major role." | ( Bardella, MT; Bufardeci, B; Colleoni, MP; Costa, B; Dasdia, T; Dolfini, E; Elli, L; Falini, ML; Floriani, I; Forlani, F; Guerrieri, N, 2002) |
| "Celiac disease is a gluten-induced enteropathy that shows a strong association with HLA-DQ2 and -DQ8." | ( Arentz-Hansen, H; Fleckenstein, B; Jung, G; Jørgensen, TJ; Lundin, KE; McAdam, SN; Molberg, Ø; Roepstorff, P; Sollid, LM, 2002) |
| "Celiac disease is a gluten-sensitive enteropathy characterized by villous atrophy that is reversed by gluten withdrawal." | ( Garrido, A; Guerrero, FJ; Lepe, JA; Muñoz, E; Ortega, C, 2002) |
| "Celiac disease is associated with impaired cholecystokinin (CCK) release." | ( Deprez, P; Geubel, A; Jouret, A; Mainguet, P; Pauwels, S; Rahier, J; Robert, A; Sempoux, C; Van Beers, BE, 2002) |
| "Celiac disease is one of the most common causes of intestinal malabsorption during childhood, and its association with insulin-dependent diabetes mellitus has been previously reported." | ( Brown, PI; Mody, RJ; Wechsler, DS, 2003) |
| "Celiac disease is a gluten-induced small bowel enteropathy." | ( Bovee-Oudenhoven, IM; De Schrijver, JE; Forget, PP; Kors, WA; Koster-Kamphuis, L; van Straaten, EA; vandDer Meer, R, 2003) |
| "Celiac disease is a chronic illness of the small bowel caused by gliadin intolerance in genetically predisposed subjects." | ( Jiskra, J; Kocna, P; Límanová, Z; Vanícková, Z, 2003) |
| "Celiac disease is thought to be a genetically based disorder reported mainly from European countries as well as countries to which Europeans have emigrated, including North America." | ( Freeman, HJ, 2003) |
| "Children with celiac disease are at risk for reduced bone mineral density." | ( Demir, H; Gürakan, F; Kavak, US; Koçak, N; Ozen, H; Saltik, IN; Yüce, A, 2003) |
| "Celiac disease is caused by T-cell responses to wheat gluten-derived peptides." | ( Bunnik, EM; de Haan, W; Drijfhout, JW; Koning, F; Kooy, YM; Stepniak, DT; Vader, LW; Van Veelen, PA, 2003) |
| "Celiac disease is a permanent intolerance to ingested gluten that results in immunologically mediated inflammatory damage of the small intestinal mucosa." | ( Mizutani, F; Oita, T; Okada, A; Sakizono, K; Tamura, A; Yamashiro, A, 2003) |
| "Celiac disease is an autoimmune disorder characterized by atrophy of the intestine villi triggered by ingestion of gluten in genetically susceptible individuals." | ( Aliberti, F; Bardella, MT; Bianchi, ML; Borghi, MO; Dubini, A; Facciuto, E; Fortunati, D; Iacomino, E; Longo, M; Migliaccio, S; Rucci, N; Saraifoger, S; Taranta, A; Teti, A, 2004) |
| "Celiac disease is associated with mineral metabolism derangement and low BMD." | ( Aliberti, F; Bardella, MT; Bianchi, ML; Borghi, MO; Dubini, A; Facciuto, E; Fortunati, D; Iacomino, E; Longo, M; Migliaccio, S; Rucci, N; Saraifoger, S; Taranta, A; Teti, A, 2004) |
| "Celiac disease is an HLA-DQ2-associated disorder characterized by intestinal T cell responses to ingested wheat gluten proteins." | ( Bergseng, E; Fleckenstein, B; Khosla, C; Molberg, Ø; Qiao, SW; Sollid, LM; Xia, J, 2004) |
| "Celiac disease is a small intestinal inflammatory disorder characterized by malabsorption, nutrient deficiency, and a range of clinical manifestations." | ( Arentz-Hansen, H; Fleckenstein, B; Jung, G; Koning, F; Lundin, KE; Molberg, Ø; Roepstorff, P; Scott, H; Sollid, LM, 2004) |
| "Celiac disease is an HLA-DQ2-associated disorder characterized by an intestinal T cell response." | ( Fleckenstein, B; Haraldsen, G; Khosla, C; Molberg, O; Oynebråten, I; Piper, J; Qiao, SW; Sollid, LM, 2005) |
| "Celiac disease is a prevalent disorder characterized by a chronic intestinal inflammation driven by HLA-DQ2 or -DQ8-restricted T cells specific for ingested wheat gluten peptides." | ( Arentz-Hansen, H; Flaete, NS; Fleckenstein, B; Jensen, T; Lundin, KE; Molberg, O; Raki, M; Sollid, LM; Uhlen, AK, 2005) |
| "Celiac disease is associated frequently with iron deficiency anemia, dermatitis herpetiformis, selective IgA deficiency, thyroid disorders, diabetes mellitus, and various connective tissue disorders but is rarely associated with cardiomyopathy." | ( Goel, NK; Kamath, PS; McBane, RD, 2005) |
| "Celiac disease is driven by intestinal T cells responsive to proline-rich gluten peptides that often harbor glutamate residues formed by tissue transglutaminase-mediated glutamine conversion." | ( Bergseng, E; Fleckenstein, B; Jung, G; Molberg, O; Qiao, SW; Sollid, LM, 2005) |
| "Celiac disease is likely a much older disorder." | ( Whittington, C, 2006) |
| "Celiac disease is caused by sensitivity to wheat gluten in genetically susceptible individuals." | ( Abuzakouk, M; Feighery, C; Jackson, J; Kelly, J; Kilmartin, C; Wieser, H, 2006) |
| "Celiac disease is another common disease in which a T-cell enteropathy is associated with increased mucosal 5-HT levels." | ( Blackshaw, E; Coleman, NS; Dunlop, SP; Foley, S; Holmes, GK; Marsden, CA; Perkins, AC; Singh, G; Spiller, RC; Wheatcroft, J, 2006) |
| "Celiac disease is associated with increased mucosal 5-HT content and enhanced 5-HT release from the upper small bowel, which correlates with postprandial dyspepsia." | ( Blackshaw, E; Coleman, NS; Dunlop, SP; Foley, S; Holmes, GK; Marsden, CA; Perkins, AC; Singh, G; Spiller, RC; Wheatcroft, J, 2006) |
| "Celiac disease is an autoimmune disorder that occurs in genetically predisposed individuals as the result of an immune response to gluten." | ( Green, PH; Hernandez, L, 2006) |
| "Celiac disease is a genetically determined bowel disease also influenced by exogenous factors in which exposure to grain components triggers a chronic immune response with intestinal symptoms." | ( Pfeiffer, C, 2006) |
| "The treatment of celiac disease is based on lifelong withdrawal of foods containing gluten." | ( Buda, A; Chiarelli, S; Curioni, A; D'Incà, R; D'Odorico, A; Martines, D; Pizzuti, D, 2006) |
| "Celiac disease is an immune-mediated enteropathy triggered by the ingestion of gluten in genetically susceptible individuals." | ( Antigoni, M; Fotoulaki, M; Karatza, E; Maria, F; Mavroudi, A; Panteliadis, C; Papastavrou, T; Spiroglou, K; Theodouli, P; Xinias, I, 2007) |
| "Celiac disease is caused by an inappropriate immune response to dietary gluten, with increased epithelial lymphocyte infiltration in the duodenum/jejunum as a hallmark." | ( Brandtzaeg, P; Farstad, IN; Jahnsen, J; Karlsson, MR; Lundin, KE; Olaussen, RW, 2007) |
| "Celiac disease is characterized by activation of HLA-DQ2/DQ8-restricted intestinal gluten-specific CD4(+) T cells." | ( Bergamo, P; Camarca, A; Costantini, S; Facchiano, AM; Gianfrani, C; Iaquinto, G; Maurano, F; Mazzarella, G; Mazzeo, MF; Rossi, M; Salvati, VM; Siciliano, RA, 2007) |
| "Celiac disease is a condition in which genetically predisposed people have an autoimmune reaction to gluten proteins found in all wheat types and closely related cereals such as barley and rye." | ( Bean, SR; Bucci, C; Caporaso, N; Ciacci, C; Del Giudice, L; Di Fonzo, N; Ioerger, B; Londei, M; Maiuri, L; Pontieri, P; Rita Massardo, D, 2007) |
| "Prevalence of celiac disease is increased in first-degree relatives with reported prevalence rates in the west between 2." | ( Aggarwal, N; Grover, R; Puri, AS; Sakhuja, P, 2007) |
| "Celiac disease is a condition in which T-cell immunity to gluten plays an important pathogenic role, yet information on DCs is scant." | ( Beattie, RM; Corazza, GR; Croft, NM; Di Sabatino, A; Gordon, JN; Leakey, NA; MacDonald, TT; Mazzarella, G; Monteleone, G; Pickard, KM; Rovedatti, L; Salvati, V; Stagg, AJ; Troncone, R; Vossenkaemper, A, 2007) |
| "Celiac disease is a gluten intolerance caused by a T-cell response against human leukocyte antigen (HLA)-DQ2 and DQ8-bound gluten peptides." | ( Blennerhassett, PA; David, CS; Huang, X; Lu, J; McKay, DM; Murray, JA; Natividad, J; Verdu, EF, 2008) |
| "Celiac disease is an intestinal disease caused by intolerance for gluten, a common protein in food." | ( Kapoerchan, VV; Koning, F; Overhand, M; Overkleeft, HS; van der Marel, GA; Wiesner, M, 2008) |
| "Celiac disease is an immune-mediated enteropathy caused by the ingestion of gluten, a protein fraction found in certain cereals." | ( Alvarez-Maqueda, M; Cebolla, A; López, MC; Manyani, H; Megías, M; Morón, B; Sousa, C; Thomas, Mdel C, 2008) |
| "Celiac disease is an enteropathy caused by intolerance to dietary gluten." | ( de Ru, AH; Drijfhout, JW; Koning, F; Moustakas, AK; Papadopoulos, GK; Stepniak, D; van Veelen, PA; Wiesner, M, 2008) |
| "Celiac disease is the most common severe food intolerance in the Western world and is due to gluten ingestion in genetically susceptible children and adults." | ( Belda, O; Garrido, A; Giráldez, A; Márquez, JL; Trigo, C; Verdejo, C, 2008) |
| "Although celiac disease is a chronic condition characterized by an intense inflammatory response of the intestinal mucosa, scant data are available about the prevalence of anemia of chronic disease in celiac disease." | ( Albertini, R; Arbustini, E; Bergamaschi, G; Biagi, F; Ciccocioppo, R; Corazza, GR; Di Sabatino, A; Markopoulos, K, 2008) |
| "Celiac disease is an autoimmune disease triggered mainly by ingestion of wheat gluten proteins." | ( Cabrera-Chávez, F; de la Barca, AM, 2009) |
| "Celiac disease is a gluten sensitive disorder that occurs in genetically susceptible individuals." | ( Borrelli, O; De Vincenzi, M; Gazza, L; Luchetti, R; Pogna, N; Silano, M; Vincentini, O, 2009) |
| "Celiac disease is a chronic inflammatory enteropathy caused by cellular immunity to dietary gluten." | ( Allison, J; Anderson, RP; Chen, Z; de Kauwe, AL; Jackson, DC; Keech, CL; Ladhams, J; McCluskey, J; Price, JD; Wijburg, O, 2009) |
| "Celiac disease is an immune-mediated disease, triggered in genetically susceptible individuals by ingested gluten from wheat, rye, barley, and other closely related cereal grains." | ( Dubois, S; Gillespie, Z; Godefroy, SB; Pulido, OM; Rashid, M; Switzer, C; Vavasour, E; Zarkadas, M, 2009) |
| "Celiac disease is an antibody-mediated enteropathy that presents permanent intolerance to ingested gluten." | ( Araújo, WM; Botelho, RB; Zandonadi, RP, 2009) |
| "Celiac disease is frequently diagnosed in patients referred for evaluation of iron deficiency anemia (IDA), being reported in 1." | ( Fernández-Bañares, F; Forné, M; Monzón, H, 2009) |
| "Individuals with celiac disease are more susceptible to pancreatic insufficiencies, dysbiosis, lactase insufficiencies, and folic acid, vitamin B12, iron, and vitamin D deficiencies, as well as accelerated bone loss due to an increase in inflammatory signaling molecules." | ( Malterre, T, 2009) |
| "Celiac disease is an enteropathy due to gluten intake in genetically predisposed individuals (HLA DQ2/DQ8)." | ( Cellier, C; Malamut, G, 2010) |
| "Celiac disease is a genetic condition that results in a debilitating immune reaction in the gut to antigens in grain." | ( Anderson, RP; Beissbarth, T; Dromey, JA; Gianfrani, C; Henderson, K; Hill, AV; Jewell, DP; Mannering, SI; McCluskey, J; Rossjohn, J; Stewart, JA; Tatham, A; Tye-Din, JA; van Heel, DA, 2010) |
| "Celiac disease is a T-cell mediated chronic inflammatory disorder of the gut that is induced by dietary exposure to gluten proteins." | ( Arntzen, MØ; Dørum, S; Fleckenstein, B; Holm, A; Koehler, CJ; Qiao, SW; Sollid, LM; Thiede, B, 2010) |
| "Celiac disease is caused by an uncontrolled immune response to gluten, a heterogeneous mixture of wheat storage proteins, including the α-gliadins." | ( Dekking, L; Drijfhout, JW; Gilissen, LJ; Goryunova, SV; Koning, F; Mitea, C; Monserrat, V; Montserrat, V; Mujico, JR; Salentijn, EM; Smulders, MJ; van den Broeck, HC; van der Meer, IM; van Veelen, P, 2010) |
| "Celiac disease is now considered to be an immune disorder that is triggered by an environmental agent in genetically predisposed persons." | ( Bucurică, S; Costache, R; Ioniţă-Radu, F; Nuţă, P; Stanciu, S, 2010) |
| "While celiac disease is estimated to affect about 1% of the world's population, it is thought to be uncommon not only in India but in Asia also." | ( Ahuja, V; Amarchand, R; Anand, K; Bhatia, V; Bhatnagar, S; Das, P; Datta Gupta, S; Goswami, A; Makharia, GK; Verma, AK, 2011) |
| "Celiac disease is more common than is recognized in India." | ( Ahuja, V; Amarchand, R; Anand, K; Bhatia, V; Bhatnagar, S; Das, P; Datta Gupta, S; Goswami, A; Makharia, GK; Verma, AK, 2011) |
| "Celiac disease is highly prevalent in patients with type 1 diabetes mellitus (11." | ( Bhadada, SK; Bhansali, A; Dutta, U; Kochhar, R; Kumar, PR; Nain, CK; Poornachandra, KS; Singh, K; Vaiphei, K, 2011) |
| "Celiac disease is a gluten-sensitive small intestinal disorder that can lead to severe villous atrophy, malabsorption, and malignancy." | ( Bors, A; Csuka, D; Czaller, I; Farkas, H; Füst, G; Kelemen, Z; Miklós, K; Molnár, K; Rajczy, K; Szabó, Z; Varga, L, 2011) |
| "Celiac disease is an autoimmune disorder that affects the gastrointestinal tract upon ingestion of gluten, which triggers the production of antibodies against gliadin and tissue transglutaminase, activating an inflammatory response and inducing tissue damage in the small intestine resulting in malabsorption." | ( Dulay, S; Iwuoha, E; Katakis, I; Lozano-Sánchez, P; O'Sullivan, CK, 2011) |
| "Celiac disease is a condition associated with the ingestion of gluten by genetically susceptible individuals." | ( Fragoso, A; O'Sullivan, CK; Ortiz, M, 2011) |
| "Celiac disease is a frequent condition that has become an important public health issue." | ( Blanco, P; Contin-Bordes, C; Guidicelli, G; Moreau, JF; Pellegrin, I; Roujon, P; Sarrat, A; Taupin, JL, 2013) |
| "Celiac disease is a chronic inflammatory condition of small intestine resulting due to sensitivity to wheat protein gluten." | ( Dhingra, D; Mantan, M; Sethi, GR, 2012) |
| "Celiac disease is a gluten-induced autoimmune enteropathy characterized by the presence of tissue tranglutaminase (tTG) autoantibodies." | ( Costa-García, A; González-García, MB; Neves, MM; Nouws, HP, 2012) |
| "Celiac disease is a diet-induced, T cell-mediated enteropathy." | ( Bodd, M; Kim, CY; Lundin, KE; Sollid, LM, 2012) |
| "Celiac disease is more common in patients with hypothyroidism." | ( Collins, D; Nathan, M; Wilcox, R; Zubarik, R, 2012) |
| "Celiac disease is a common and lifelong food intolerance, affecting approximately 1% of the population." | ( Camarca, A; Del Mastro, A; Gianfrani, C, 2012) |
| "Although celiac disease is the most prevalent bowel abnormality associated with intestinal malabsorption, its relationship to high kidney oxalate burden and decreased kidney function has not been established." | ( Abul-Ezz, S; Capolongo, G; Moe, OW; Sakhaee, K, 2012) |
| "Celiac disease is an enteropathy triggered by dietary gluten found in wheat, barley, and rye." | ( Ciclitira, PJ; Ellis, HJ; Herencia, LI; Suligoj, T; Zevallos, VF, 2012) |
| "Celiac disease is an immune-mediated enteropathy, characterized by lifelong intolerance to gluten in genetically susceptible individuals." | ( Prabhasankar, P; Susanna, S, 2012) |
| "Celiac disease is a genetically-based and immunologically-mediated systemic disorder, affecting primarily the small intestine in adults." | ( Freeman, HJ, 2012) |
| "Celiac disease is a chronic enteropathy caused by the ingestion of wheat gliadin and other cereal prolamines." | ( Addeo, F; D'Auria, S; Ferranti, P; Fierro, O; Iacomino, G; Liguori, C; Mamone, G; Picariello, G, 2013) |
| "Celiac disease is an immune-mediated enteropathy caused by a permanent sensitivity to gluten in genetically susceptible individuals." | ( Ertekin, V; Küçük, N; Tozun, MS, 2013) |
| "Celiac disease is an autoimmune disorder resulting from gluten intolerance and is based on a genetically predisposition." | ( Hahn, A; Ströhle, A; Wolters, M, 2013) |
| "Celiac disease is emerging in India and has become a public health problem." | ( Makharia, GK; Rajpoot, P, 2013) |
| "Celiac disease is an autoimmune disease that affects the villi of the small intestine causing abdominal pain, gas, diarrhea, or bad absorption due to gluten intolerance." | ( Burló, F; Carbonell-Barrachina, AA; Munera-Picazo, S; Ramírez-Gandolfo, A, 2014) |
| "Celiac disease is a food-induced enteropathy resulting from exposure to gluten in genetically predisposed individuals." | ( de Lorgeril, M; Salen, P, 2014) |
| "Celiac disease is a food-sensitive enteropathy triggered by the ingestion of wheat gluten proteins and related proteins from barley, rye, and some varieties of oat." | ( Barro, F; Gil-Humanes, J; Pistón, F; Rosell, CM, 2014) |
| "Celiac disease is a T cell-mediated disease induced by dietary gluten, a component of which is gliadin." | ( Anderson, RP; Beringer, DX; Drijfhout, JW; Kan, WT; Koning, F; Kooy-Winkelaar, Y; La Gruta, NL; Loh, KL; Mearin, ML; Montserrat, V; Mujico, JR; Petersen, J; Reid, HH; Rossjohn, J; Schweizer, J; Thompson, A; van Bergen, J; van Lummel, M, 2014) |
| "Celiac disease is an immune-mediated disorder triggered by gluten in genetically susceptible persons." | ( Catassi, C; Lionetti, E, 2014) |
| "Celiac disease is an autoimmune enteropathy triggered by the ingestion of gluten, characterized by immune responses toward gluten constituents and the autoantigen transglutaminase 2." | ( Caminero, A; McCarville, JL; Verdu, EF, 2015) |
| "Celiac disease is an autoimmune enteropathy caused by a permanent intolerance to dietary gluten in genetically predisposed individuals." | ( Liu, M; Portincasa, P; Tso, P; Wang, DQ; Wang, HH, 2016) |
| "Celiac disease is an autoimmune disease of the small intestine which occurs in genetically predisposed people of all ages." | ( Drosou, ME; Fontalis, A; Giouleme, O; Hadwe, SE; Lazaridis, A; Petidis, K; Prousali, E, 2016) |
| "Celiac disease is considered to be rare in parts of the world where rice is a staple cereal." | ( Khajuria, A; Pandita, KK; Rafiq, A; Razdan, S, 2016) |
| "Initiation of celiac disease is triggered in the gastrointestinal tract by transglutaminase 2 (TG2) assisted deamidation of gluten peptides." | ( Engstrom, N; Saenz-Méndez, P; Scheers, J; Scheers, N, 2017) |
| "Celiac disease is a chronic immune-mediated inflammatory disorder of the gut triggered by dietary gluten." | ( Anderson, RP; Cook, L; Gillies, JK; Hardy, MY; Kelleher, AD; Levings, MK; Munier, CML; Ontiveros, N; Petersen, J; Reid, HH; Rossjohn, J; Seddiki, N; Tye-Din, JA; van Bockel, D; Zaunders, JJ, 2017) |
| "Celiac disease is among the most important health problems and the most important feature of this disease is its being the only lifelong food allergy." | ( Demir, MK; Kutlu, G; Yilmaz, MT, 2017) |
| "Celiac disease is an autoimmune disorder due to the inflammatory response to gluten in genetically predisposed individuals." | ( Adams, DW; Naik, RD; Seidner, DL, 2018) |
| "Celiac disease is a chronic inflammation of the small intestine with villous atrophy that can become refractory to a gluten-free diet." | ( Alcantara, M; Brousse, N; Bruneau, J; Cellier, C; Cerf-Bensussan, N; Derrieux, C; Hermine, O; Lhermitte, L; Macintyre, E; Malamut, G; Meresse, B; Molina, T; Sibon, D; Trinquand, A; Verkarre, V; Villarese, P, 2019) |
| "Celiac disease is an autoimmune disorder that develops because of sensitivity to gluten-containing grains in genetically disposed individuals." | ( Atan, D; Comba, A, 2020) |
| "Celiac disease is an autoimmune illness activated by gluten peptides produced during gastrointestinal digestion." | ( Demarais, N; Domigan, L; Gerrard, J; Larsen, N; Ogilvie, O; Roberts, S; Sutton, K, 2020) |
| "Celiac disease is an autoimmune enteropathy characterized by an aberrant immune response to ingested gluten in genetically predisposed individuals." | ( Abrams, JA; Alaedini, A; Freedberg, DE; Green, PHR; Jang, S; Lebwohl, B, 2020) |
| "Celiac disease is activated by digestion-resistant gluten peptides that contain immunogenic epitopes." | ( Domigan, LJ; Gerrard, JA; Larsen, N; Ogilvie, OJ; Roberts, S; Sutton, KH, 2021) |
| "Refractory celiac disease is an uncommon condition which might be associated to poor prognosis." | ( Pêgas, KL; Salgado, K; Soldera, J, 2021) |
| "Celiac disease is the most common chronic gastroenterological disease." | ( Kovačić, I; Kovačić, M; Veršić, M, 2021) |
| "Celiac disease is a multisystem immune based disorder, caused by an immune-mediated reaction to ingested gluten with increasing prevalence in the USA." | ( Du, Z; Li, L; Li, Z; Zhang, L, 2023) |
| "Celiac disease is an autoimmune-mediated chronic inflammatory condition of the small intestine affecting about 1-2% of people in Caucasian populations." | ( Aeschlimann, D; Büchold, C; Gerlach, U; Heil, A; Hils, M; Pasternack, R; Pelzer, C; Weber, J, 2022) |
| "Type 1 refractory celiac disease is characterized by a normal intraepithelial lymphocyte population and type 2 is defined by the presence of an aberrant, clonal intraepithelial lymphocyte population." | ( Green, PHR; Ko, CW; Paski, S; Rubio-Tapia, A, 2022) |
| "Celiac disease is therefore one of the intestinal diseases that can lead to hyperoxaluria and oxalate nephropathy." | ( Stegeman, CA; Zijlstra, HW, 2023) |
| Excerpt | Reference |
|---|
| "In untreated celiac disease presenting with subtotal villous atrophy, peak concentrations of primary BAs were significantly delayed and serum levels were significantly higher than in controls." | ( Ejderhamn, J; Samuelson, K; Strandvik, B, 1992) |
| "In patients with untreated celiac disease half-times were 154." | ( Chadwick, VS; Fitzpatrick, ML; Maton, PN; Selden, AC, 1985) |
| "It was concluded that the mucosa in untreated celiac disease was more "leaky" than normal." | ( Cooper, BT; Ukabam, SO, 1984) |
| "In the 14 children with untreated celiac disease both basal [81 (SD 33) pmol/L] and postprandial [129 (SD 26) pmol/L] concentrations of enteroglucagon were significantly higher (p less than 0." | ( Kilander, AF; Lindstedt, G; Lundberg, PA; Stenhammar, L, 1984) |
| "Only early diagnosis and treatment of celiac disease can avoid the deterioration of the bone structure observed in all clinical status of celiac disease." | ( Bai, JC; Boerr, L; Gonzalez, D; Mautalen, C; Mazure, R; Pedreira, S; Vazquez, H, 1994) |
| "Serum from 53 children with untreated celiac disease, 22 in remission and 13 on challenge, were screened for antigliadin IgA, antigliadin IgG, and IgA reticulin antibodies, in addition to IgA endomysium antibodies tested both on monkey esophagus and on human umbilical cord." | ( Kolho, KL; Savilahti, E, 1997) |
| "Vitamin B12 deficiency is common in untreated celiac disease, and concentrations should be measured routinely before hematinic replacement." | ( Dahele, A; Ghosh, S, 2001) |
| "We conclude that basal PYY levels in untreated celiac disease are elevated, that predigestion of fat enhances PYY release in these patients, and that PYY secretion is correlated with CCK release." | ( Hopman, WP; Jansen, JB; Wahab, PJ, 2001) |
| "In 25 patients with untreated celiac disease and 23 sex- and age-matched healthy volunteers, breath H(2) excretion was measured after ingestion of a 250-ml solution containing sorbitol, a poorly absorbed alcohol sugar." | ( Corazza, GR; Di Stefano, M; Malservisi, S; Miceli, E; Missanelli, A; Strocchi, A, 2003) |
| "Early diagnosis and treatment of celiac disease during childhood will protect the patient from osteoporosis." | ( Demir, H; Gürakan, F; Kavak, US; Koçak, N; Ozen, H; Saltik, IN; Yüce, A, 2003) |
| "Twenty-four adult females with treated celiac disease and twenty age- and sex-matched control subjects were studied." | ( Ang, L; Butcher, GP; Collins, C; Finch, PJ; Heaney, RP; Maxwell, JD; Pazianas, M; Subhani, JM; Zaidi, M, 2005) |
| "In Italian patients with untreated celiac disease, HFE mutations do not constitute a protective factor against the development of iron deficiency, which seems to be mainly determined by the severity of the intestinal lesions." | ( Bardella, MT; Barisani, D; Ceroni, S; Del Bianco, S; Meneveri, R, 2004) |
| "L-Carnitine blood levels are low in untreated celiac disease." | ( Bucci, C; Calvani, M; Ciacci, C; Iannoni, E; Iovino, P; Margarucci, S; Peluso, G; Rispo, A; Siniscalchi, M; Tortora, R; Zingone, F, 2007) |
| "Our results show that the patients of untreated celiac disease should be instructed not to eat things made up of wheat one day before hydrogen breath testing so that the normal fasting hydrogen concentration can be obtained and false-negative hydrogen breath test results can be avoided." | ( Kaur, J; Prasad, KK; Rana, SV; Sharma, S; Singh, K; Sinha, SK, 2009) |
| "Currently the only treatment for celiac disease is a lifelong gluten-free diet." | ( Auriola, S; Jantunen, A; Kaukinen, K; Kaukovirta-Norja, A; Laurila, K; Lindfors, K; Mäki, M; Männisto, PT; Mauriala, T; Qiao, SW; Sollid, LM; Stenman, SM; Venäläinen, JI, 2009) |
| "The only treatment for celiac disease is a strict gluten-free diet for life." | ( Dubois, S; Gillespie, Z; Godefroy, SB; Pulido, OM; Rashid, M; Switzer, C; Vavasour, E; Zarkadas, M, 2009) |
| "The osteoporosis did not respond to the treatment of his celiac disease but was completely reversed by bisphosphonate therapy." | ( Determann, C; Fraser, WD; McRorie, E; Ralston, SH; Riches, PL; van't Hof, R, 2009) |
| "Sixty consecutive patients with untreated celiac disease, 45 patients with celiac disease treated with a gluten-free diet for at least 1 year, and 30 control subjects were enrolled in this study." | ( Di Tola, M; Donato, G; Gualdi, G; Libanori, V; Masselli, G; Palumbo, P; Picarelli, A; Piermattei, A; Pittalis, A; Polettini, E; Saponara, A, 2010) |
| "A gluten-free diet is the basis of treatment of celiac disease, but the role of an elimination diet in eosinophilic esophagitis is not well established." | ( Escudero, C; Fernández-Rodríguez, M; Ibáñez, MD; Martinez-Gómez, MJ; Rodríguez del Río, P; Sánchez-García, S; Vereda, A, 2011) |
| "Doses decreased significantly after treatment of celiac disease (154 μg vs 111 μg, P=." | ( Collins, D; Nathan, M; Wilcox, R; Zubarik, R, 2012) |
| "In patients with treated celiac disease (CD), the ingestion of gluten traces contained in gluten-free (GF) wheat substitutes (eg, GF bread, flour, and pasta) could cause persisting intestinal mucosal damage." | ( Canela, MA; Catassi, C; Fasano, A; Gibert, A; Houben, GF; Kruizinga, AG; Neuhold, S, 2013) |
| "The current treatment for celiac disease is strict gluten-free diet." | ( Andersson, LC; Freitag, TL; Loponen, J; Meri, S; Messing, M; Saavalainen, P; Salovaara, H; Schuppan, D; Sontag-Strohm, T; Zevallos, V, 2014) |
| "This case affirms that chronic untreated celiac disease, can lead to an important bone loss and irreversible complications like skeletal deformities." | ( Abourazzak, F; Azzouzi, H; Harzy, T; Squalli, G; Tahiri, L, 2014) |
| "The only treatment for celiac disease is the gluten-free diet." | ( Campos, Tdo N; Cardoso, Lde M; Gilberti, FA; Hermsdorff, HH; Moraes, LF; Peluzio, Mdo C; Pinheiro-Sant'Ana, HM; Valente, FX, 2015) |
| "The molecule was designed for the treatment of celiac disease." | ( Aeschlimann, D; Büchold, C; Gerlach, U; Heil, A; Hils, M; Pasternack, R; Pelzer, C; Weber, J, 2022) |
| "Children with untreated celiac disease have lower expression of genes important for IGN." | ( Agardh, D; Arnell, H; Gudjonsdottir, AH; Karlson, O; Torinsson Naluai, Å, 2022) |