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3',5'-dimethylacetaminophen

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Description

3',5'-dimethylacetaminophen: structure given in first source [Medical Subject Headings (MeSH), National Library of Medicine, extracted Dec-2023]

Cross-References

ID SourceID
PubMed CID89896
CHEMBL ID278698
SCHEMBL ID2552398
MeSH IDM0125546

Synonyms (18)

Synonym
3',5'-dimethylacetaminophen
acetamide, n-(4-hydroxy-3,5-dimethylphenyl)-
3,5-dimethyl-4-hydroxyacetanilide
acetamide, n-(3,5-dimethyl-4-hydroxyphenyl)-
3',5'-acetoxylidide, 4'-hydroxy-
3,5-dimethylparacetamol
3,5-dimethylacetaminophen
brn 2723443
22900-79-4
CHEMBL278698
n-(4-hydroxy-3,5-dimethylphenyl)acetamide
3',5'-dimethyl-4'-hydroxyacetanilide
3',5'-dimethylparacetamol
FQCIYRLHNCRDKD-UHFFFAOYSA-N
SCHEMBL2552398
DTXSID00177443
3',5'-di-methyl-4'-hydroxyacetanilide
Z1509144456
[information is derived through text-mining from research data collected from National Library of Medicine (NLM), extracted Dec-2023]

Bioassays (30)

Assay IDTitleYearJournalArticle
AID191477Nephrotoxicity upon intragastric administration was assessed in rat liver as centrilobular vacuolation or necrosis at a dose of 5 mM1980Journal of medicinal chemistry, Nov, Volume: 23, Issue:11
Studies on the mechanism of toxicity of acetaminophen. Synthesis and reactions of N-acetyl-2,6-dimethyl- and N-acetyl-3,5-dimethyl-p-benzoquinone imines.
AID120613Nephrotoxicity upon intragastric administration was assessed in mice liver as centrilobular vacuolation or necrosis at a dose of 5 mM1980Journal of medicinal chemistry, Nov, Volume: 23, Issue:11
Studies on the mechanism of toxicity of acetaminophen. Synthesis and reactions of N-acetyl-2,6-dimethyl- and N-acetyl-3,5-dimethyl-p-benzoquinone imines.
AID191487Nephrotoxicity upon intravenous administration was assessed in rat kidney as proximal tublar necrosis deep in the cortex at a dose of 10 mM1980Journal of medicinal chemistry, Nov, Volume: 23, Issue:11
Studies on the mechanism of toxicity of acetaminophen. Synthesis and reactions of N-acetyl-2,6-dimethyl- and N-acetyl-3,5-dimethyl-p-benzoquinone imines.
AID123230Nephrotoxicity upon intraperitoneal administration was assessed in mice liver as centrilobular vacuolation or necrosis at a dose of 2 mM1980Journal of medicinal chemistry, Nov, Volume: 23, Issue:11
Studies on the mechanism of toxicity of acetaminophen. Synthesis and reactions of N-acetyl-2,6-dimethyl- and N-acetyl-3,5-dimethyl-p-benzoquinone imines.
AID120616Nephrotoxicity upon intraperitoneal administration was assessed in mice kidney as proximal tublar necrosis deep in the cortex at a dose of 2 mM1980Journal of medicinal chemistry, Nov, Volume: 23, Issue:11
Studies on the mechanism of toxicity of acetaminophen. Synthesis and reactions of N-acetyl-2,6-dimethyl- and N-acetyl-3,5-dimethyl-p-benzoquinone imines.
AID120611Nephrotoxicity upon intragastric administration was assessed in mice liver as centrilobular vacuolation or necrosis at a dose of 10 mM1980Journal of medicinal chemistry, Nov, Volume: 23, Issue:11
Studies on the mechanism of toxicity of acetaminophen. Synthesis and reactions of N-acetyl-2,6-dimethyl- and N-acetyl-3,5-dimethyl-p-benzoquinone imines.
AID123231Nephrotoxicity upon intraperitoneal administration was assessed in mice liver as centrilobular vacuolation or necrosis at a dose of 5 mM1980Journal of medicinal chemistry, Nov, Volume: 23, Issue:11
Studies on the mechanism of toxicity of acetaminophen. Synthesis and reactions of N-acetyl-2,6-dimethyl- and N-acetyl-3,5-dimethyl-p-benzoquinone imines.
AID191484Nephrotoxicity upon intraperitoneal administration was assessed in rat liver as centrilobular vacuolation or necrosis at a dose of 2 mM1980Journal of medicinal chemistry, Nov, Volume: 23, Issue:11
Studies on the mechanism of toxicity of acetaminophen. Synthesis and reactions of N-acetyl-2,6-dimethyl- and N-acetyl-3,5-dimethyl-p-benzoquinone imines.
AID120608Nephrotoxicity upon intragastric administration was assessed in mice kidney as proximal tublar necrosis deep in the cortex at a dose of 2 mM1980Journal of medicinal chemistry, Nov, Volume: 23, Issue:11
Studies on the mechanism of toxicity of acetaminophen. Synthesis and reactions of N-acetyl-2,6-dimethyl- and N-acetyl-3,5-dimethyl-p-benzoquinone imines.
AID191491Nephrotoxicity upon intravenous administration was assessed in rat liver as centrilobular vacuolation or necrosis at a dose of 10 mM1980Journal of medicinal chemistry, Nov, Volume: 23, Issue:11
Studies on the mechanism of toxicity of acetaminophen. Synthesis and reactions of N-acetyl-2,6-dimethyl- and N-acetyl-3,5-dimethyl-p-benzoquinone imines.
AID191483Nephrotoxicity upon intraperitoneal administration was assessed in rat liver as centrilobular vacuolation or necrosis at a dose of 10 mM1980Journal of medicinal chemistry, Nov, Volume: 23, Issue:11
Studies on the mechanism of toxicity of acetaminophen. Synthesis and reactions of N-acetyl-2,6-dimethyl- and N-acetyl-3,5-dimethyl-p-benzoquinone imines.
AID120615Nephrotoxicity upon intraperitoneal administration was assessed in mice kidney as proximal tublar necrosis deep in the cortex at a dose of 10 mM1980Journal of medicinal chemistry, Nov, Volume: 23, Issue:11
Studies on the mechanism of toxicity of acetaminophen. Synthesis and reactions of N-acetyl-2,6-dimethyl- and N-acetyl-3,5-dimethyl-p-benzoquinone imines.
AID191476Nephrotoxicity upon intragastric administration was assessed in rat liver as centrilobular vacuolation or necrosis at a dose of 2 mM1980Journal of medicinal chemistry, Nov, Volume: 23, Issue:11
Studies on the mechanism of toxicity of acetaminophen. Synthesis and reactions of N-acetyl-2,6-dimethyl- and N-acetyl-3,5-dimethyl-p-benzoquinone imines.
AID120619Nephrotoxicity upon intraperitoneal administration was assessed in mice liver as centrilobular vacuolation or necrosis at a dose of 10 mM1980Journal of medicinal chemistry, Nov, Volume: 23, Issue:11
Studies on the mechanism of toxicity of acetaminophen. Synthesis and reactions of N-acetyl-2,6-dimethyl- and N-acetyl-3,5-dimethyl-p-benzoquinone imines.
AID191480Nephrotoxicity upon intraperitoneal administration was assessed in rat kidney as proximal tublar necrosis deep in the cortex at a dose of 2 mM1980Journal of medicinal chemistry, Nov, Volume: 23, Issue:11
Studies on the mechanism of toxicity of acetaminophen. Synthesis and reactions of N-acetyl-2,6-dimethyl- and N-acetyl-3,5-dimethyl-p-benzoquinone imines.
AID191493Nephrotoxicity upon intravenous administration was assessed in rat liver as centrilobular vacuolation or necrosis at a dose of 5 mM1980Journal of medicinal chemistry, Nov, Volume: 23, Issue:11
Studies on the mechanism of toxicity of acetaminophen. Synthesis and reactions of N-acetyl-2,6-dimethyl- and N-acetyl-3,5-dimethyl-p-benzoquinone imines.
AID191472Nephrotoxicity upon intragastric administration was assessed in rat kidney as proximal tublar necrosis deep in the cortex at a dose of 2 mM1980Journal of medicinal chemistry, Nov, Volume: 23, Issue:11
Studies on the mechanism of toxicity of acetaminophen. Synthesis and reactions of N-acetyl-2,6-dimethyl- and N-acetyl-3,5-dimethyl-p-benzoquinone imines.
AID191481Nephrotoxicity upon intraperitoneal administration was assessed in rat kidney as proximal tublar necrosis deep in the cortex at a dose of 5 mM1980Journal of medicinal chemistry, Nov, Volume: 23, Issue:11
Studies on the mechanism of toxicity of acetaminophen. Synthesis and reactions of N-acetyl-2,6-dimethyl- and N-acetyl-3,5-dimethyl-p-benzoquinone imines.
AID191479Nephrotoxicity upon intraperitoneal administration was assessed in rat kidney as proximal tublar necrosis deep in the cortex at a dose of 10 mM1980Journal of medicinal chemistry, Nov, Volume: 23, Issue:11
Studies on the mechanism of toxicity of acetaminophen. Synthesis and reactions of N-acetyl-2,6-dimethyl- and N-acetyl-3,5-dimethyl-p-benzoquinone imines.
AID191475Nephrotoxicity upon intragastric administration was assessed in rat liver as centrilobular vacuolation or necrosis at a dose of 10 mM1980Journal of medicinal chemistry, Nov, Volume: 23, Issue:11
Studies on the mechanism of toxicity of acetaminophen. Synthesis and reactions of N-acetyl-2,6-dimethyl- and N-acetyl-3,5-dimethyl-p-benzoquinone imines.
AID120612Nephrotoxicity upon intragastric administration was assessed in mice liver as centrilobular vacuolation or necrosis at a dose of 2 mM1980Journal of medicinal chemistry, Nov, Volume: 23, Issue:11
Studies on the mechanism of toxicity of acetaminophen. Synthesis and reactions of N-acetyl-2,6-dimethyl- and N-acetyl-3,5-dimethyl-p-benzoquinone imines.
AID120617Nephrotoxicity upon intraperitoneal administration was assessed in mice kidney as proximal tublar necrosis deep in the cortex at a dose of 5 mM1980Journal of medicinal chemistry, Nov, Volume: 23, Issue:11
Studies on the mechanism of toxicity of acetaminophen. Synthesis and reactions of N-acetyl-2,6-dimethyl- and N-acetyl-3,5-dimethyl-p-benzoquinone imines.
AID191473Nephrotoxicity upon intragastric administration was assessed in rat kidney as proximal tublar necrosis deep in the cortex at a dose of 5 mM1980Journal of medicinal chemistry, Nov, Volume: 23, Issue:11
Studies on the mechanism of toxicity of acetaminophen. Synthesis and reactions of N-acetyl-2,6-dimethyl- and N-acetyl-3,5-dimethyl-p-benzoquinone imines.
AID120609Nephrotoxicity upon intragastric administration was assessed in mice kidney as proximal tublar necrosis deep in the cortex at a dose of 5 mM1980Journal of medicinal chemistry, Nov, Volume: 23, Issue:11
Studies on the mechanism of toxicity of acetaminophen. Synthesis and reactions of N-acetyl-2,6-dimethyl- and N-acetyl-3,5-dimethyl-p-benzoquinone imines.
AID191485Nephrotoxicity upon intraperitoneal administration was assessed in rat liver as centrilobular vacuolation or necrosis at a dose of 5 mM1980Journal of medicinal chemistry, Nov, Volume: 23, Issue:11
Studies on the mechanism of toxicity of acetaminophen. Synthesis and reactions of N-acetyl-2,6-dimethyl- and N-acetyl-3,5-dimethyl-p-benzoquinone imines.
AID191489Nephrotoxicity upon intravenous administration was assessed in rat kidney as proximal tublar necrosis deep in the cortex at a dose of 5 mM1980Journal of medicinal chemistry, Nov, Volume: 23, Issue:11
Studies on the mechanism of toxicity of acetaminophen. Synthesis and reactions of N-acetyl-2,6-dimethyl- and N-acetyl-3,5-dimethyl-p-benzoquinone imines.
AID191471Nephrotoxicity upon intragastric administration was assessed in rat kidney as proximal tublar necrosis deep in the cortex at a dose of 10 mM1980Journal of medicinal chemistry, Nov, Volume: 23, Issue:11
Studies on the mechanism of toxicity of acetaminophen. Synthesis and reactions of N-acetyl-2,6-dimethyl- and N-acetyl-3,5-dimethyl-p-benzoquinone imines.
AID191492Nephrotoxicity upon intravenous administration was assessed in rat liver as centrilobular vacuolation or necrosis at a dose of 2 mM1980Journal of medicinal chemistry, Nov, Volume: 23, Issue:11
Studies on the mechanism of toxicity of acetaminophen. Synthesis and reactions of N-acetyl-2,6-dimethyl- and N-acetyl-3,5-dimethyl-p-benzoquinone imines.
AID120607Nephrotoxicity upon intragastric administration was assessed in mice kidney as proximal tublar necrosis deep in the cortex at a dose of 10 mM1980Journal of medicinal chemistry, Nov, Volume: 23, Issue:11
Studies on the mechanism of toxicity of acetaminophen. Synthesis and reactions of N-acetyl-2,6-dimethyl- and N-acetyl-3,5-dimethyl-p-benzoquinone imines.
AID191488Nephrotoxicity upon intravenous administration was assessed in rat kidney as proximal tublar necrosis deep in the cortex at a dose of 2 mM1980Journal of medicinal chemistry, Nov, Volume: 23, Issue:11
Studies on the mechanism of toxicity of acetaminophen. Synthesis and reactions of N-acetyl-2,6-dimethyl- and N-acetyl-3,5-dimethyl-p-benzoquinone imines.
[information is prepared from bioassay data collected from National Library of Medicine (NLM), extracted Dec-2023]

Research

Studies (9)

TimeframeStudies, This Drug (%)All Drugs %
pre-19906 (66.67)18.7374
1990's3 (33.33)18.2507
2000's0 (0.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80
[information is prepared from research data collected from National Library of Medicine (NLM), extracted Dec-2023]

Market Indicators

Research Demand Index: 12.05

According to the monthly volume, diversity, and competition of internet searches for this compound, as well the volume and growth of publications, there is estimated to be weak demand-to-supply ratio for research on this compound.

MetricThis Compound (vs All)
Research Demand Index12.05 (24.57)
Research Supply Index2.30 (2.92)
Research Growth Index4.36 (4.65)
Search Engine Demand Index0.00 (26.88)
Search Engine Supply Index0.00 (0.95)

This Compound (12.05)

All Compounds (24.57)

Study Types

Publication TypeThis drug (%)All Drugs (%)
Trials0 (0.00%)5.53%
Reviews1 (11.11%)6.00%
Case Studies0 (0.00%)4.05%
Observational0 (0.00%)0.25%
Other8 (88.89%)84.16%
[information is prepared from research data collected from National Library of Medicine (NLM), extracted Dec-2023]